Vacuolin-1 inhibits autophagy by impairing lysosomal maturation via PIKfyve inhibition

FEBS Lett. 2016 Jun;590(11):1576-85. doi: 10.1002/1873-3468.12195. Epub 2016 May 13.

Abstract

Lysosomal protein degradation via autophagy strictly regulates cellular protein homoeostasis. Herein we performed high-content screening to identify compounds that inhibit autophagy pathways. We obtained 11 hit compounds and performed cluster analysis using cellular morphological information. Vacuolin-1, which induces the formation of giant vacuoles and is a target unknown compound, clustered with the known PIKfyve inhibitor YM201636. We further confirmed that vacuolin-1 is a potent PIKfyve inhibitor, and we finally concluded that PIKfyve inhibitors are novel chemical tools for regulating autophagy.

Keywords: PIKfyve; autophagy; vacuolin-1.

MeSH terms

  • Autophagy / drug effects*
  • Cells, Cultured
  • Drug Discovery / methods
  • Drug Screening Assays, Antitumor / methods
  • HeLa Cells
  • Heterocyclic Compounds, 4 or More Rings / pharmacology*
  • High-Throughput Screening Assays
  • Humans
  • Lysosomes / drug effects*
  • Lysosomes / metabolism
  • Phosphatidylinositol 3-Kinases
  • Phosphoinositide-3 Kinase Inhibitors*
  • Protein Kinase Inhibitors / pharmacology

Substances

  • Heterocyclic Compounds, 4 or More Rings
  • Phosphoinositide-3 Kinase Inhibitors
  • Protein Kinase Inhibitors
  • vacuolin-1
  • PIKFYVE protein, human