ATR-mediated regulation of nuclear and cellular plasticity

DNA Repair (Amst). 2016 Aug:44:143-150. doi: 10.1016/j.dnarep.2016.05.020. Epub 2016 May 16.

Abstract

ATR (Ataxia Telangiectasia and Rad3-related) is a member of the Phosphatidylinositol 3-kinase-related kinases (PIKKs) family, amongst six other vertebrate proteins known so far. ATR is indispensable for cell survival and its essential role is in sensing DNA damage and initiating appropriate repair responses. In this review we highlight emerging and recent observations connecting ATR to alternative roles in controlling the nuclear envelope, nucleolus, centrosome and other organelles in response to both internal and external stress conditions. We propose that ATR functions control cell plasticity by sensing structural deformations of different cellular components, including DNA and initiating appropriate repair responses, most of which are yet to be understood completely.

Keywords: ATR; Cellular plasticity; Centrosome; Nuclear envelope; Nucleolus; PIKK.

Publication types

  • Review
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Ataxia Telangiectasia Mutated Proteins / genetics
  • Ataxia Telangiectasia Mutated Proteins / metabolism
  • Cell Nucleolus / metabolism
  • Cell Nucleolus / ultrastructure
  • Cell Plasticity*
  • Centrosome / metabolism
  • Centrosome / ultrastructure
  • DNA / chemistry
  • DNA / metabolism*
  • DNA Damage
  • DNA Repair*
  • Eukaryotic Cells / metabolism
  • Eukaryotic Cells / ultrastructure
  • Humans
  • Nuclear Envelope / metabolism
  • Nuclear Envelope / ultrastructure

Substances

  • DNA
  • ATR protein, human
  • Ataxia Telangiectasia Mutated Proteins