Reconsolidation-induced memory persistence: Participation of late phase hippocampal ERK activation

Neurobiol Learn Mem. 2016 Sep:133:79-88. doi: 10.1016/j.nlm.2016.06.013. Epub 2016 Jun 15.

Abstract

Persistence is an attribute of long-term memories (LTM) that has recently caught researcher's attention in search for mechanisms triggered by experience that assure memory perdurability. Up-to-date, scarce evidence of relationship between reconsolidation and persistence has been described. Here, we characterized hippocampal ERK participation in LTM reconsolidation and persistence using an inhibitory avoidance task (IA) at different time points. Intra-dorsal-hippocampal (dHIP) administration of an ERK inhibitor (PD098059, PD, 1.0μg/hippocampus) 3h after retrieval did not affect reconsolidation of a strong IA, when tested 24h apart. However, the same manipulation impaired performance when animals were tested at 7d, regardless of the training's strength; and being specific to memory reactivation. To the best of our knowledge, this is the first report showing that persistence might be triggered after memory reactivation involving an ERK/MAPK-dependent process.

Keywords: ERK/MAPK; Inhibitory avoidance; Memory persistence; Memory reconsolidation; Mice; PD098059.

MeSH terms

  • Animals
  • Avoidance Learning / drug effects
  • Avoidance Learning / physiology*
  • Extracellular Signal-Regulated MAP Kinases / antagonists & inhibitors
  • Extracellular Signal-Regulated MAP Kinases / metabolism*
  • Flavonoids / administration & dosage
  • Flavonoids / pharmacology
  • Hippocampus / drug effects
  • Hippocampus / metabolism*
  • Memory Consolidation / drug effects
  • Memory Consolidation / physiology*
  • Memory, Long-Term / drug effects
  • Memory, Long-Term / physiology*
  • Mental Recall / drug effects
  • Mental Recall / physiology*
  • Mice
  • Protein Kinase Inhibitors / administration & dosage
  • Protein Kinase Inhibitors / pharmacology*
  • Time Factors

Substances

  • Flavonoids
  • Protein Kinase Inhibitors
  • Extracellular Signal-Regulated MAP Kinases
  • 2-(2-amino-3-methoxyphenyl)-4H-1-benzopyran-4-one