Bile acids in regulation of inflammation and immunity: friend or foe?

Clin Exp Rheumatol. 2016 Jul-Aug;34(4 Suppl 98):25-31. Epub 2016 Jul 29.

Abstract

Apart from their pivotal role in dietary lipid absorption and cholesterol homeostasis, bile acids (BAs) are increasingly recognised as important signalling molecules in the regulation of systemic endocrine functions. As such BAs are natural ligands for several nuclear hormone receptors and G-protein-coupled receptors. Through activating various signalling pathways, BAs not only regulate their own synthesis, enterohepatic recirculation and metabolism, but also immune homeostasis. This makes BAs attractive therapeutic agents for managing metabolic and inflammatory liver disorders. Recent experimental and clinical evidence indicates that BAs exert beneficial effects in cholestatic and metabolically driven inflammatory diseases. This review elucidates how different BAs function as pathogenetic factors and potential therapeutic agents for inflammation-driven liver diseases, focusing on their role in regulation of inflammation and immunity.

Publication types

  • Review

MeSH terms

  • Adaptive Immunity
  • Animals
  • Anti-Inflammatory Agents / therapeutic use
  • Bile Acids and Salts / immunology*
  • Bile Acids and Salts / metabolism
  • Bile Acids and Salts / therapeutic use
  • Bile Ducts / immunology
  • Bile Ducts / metabolism
  • Bile Ducts / pathology
  • Humans
  • Immunity, Innate
  • Inflammation / immunology*
  • Inflammation / metabolism
  • Inflammation / pathology
  • Inflammation / prevention & control
  • Inflammation Mediators / immunology
  • Inflammation Mediators / metabolism
  • Ligands
  • Liver / immunology
  • Liver / metabolism
  • Liver / pathology
  • Receptors, Cytoplasmic and Nuclear / agonists
  • Receptors, Cytoplasmic and Nuclear / metabolism
  • Receptors, G-Protein-Coupled / agonists
  • Receptors, G-Protein-Coupled / metabolism
  • Signal Transduction

Substances

  • Anti-Inflammatory Agents
  • Bile Acids and Salts
  • GPBAR1 protein, human
  • Inflammation Mediators
  • Ligands
  • Receptors, Cytoplasmic and Nuclear
  • Receptors, G-Protein-Coupled
  • farnesoid X-activated receptor