The impact of Roux-en-Y gastric bypass surgery on normal metabolism in a porcine model

PLoS One. 2017 Mar 3;12(3):e0173137. doi: 10.1371/journal.pone.0173137. eCollection 2017.

Abstract

Background: A growing body of literature on Roux-en-Y gastric bypass surgery (RYGB) has generated inconclusive results on the mechanism underlying the beneficial effects on weight loss and glycaemia, partially due to the problems of designing clinical studies with the appropriate controls. Moreover, RYGB is only performed in obese individuals, in whom metabolism is perturbed and not completely understood.

Methods: In an attempt to isolate the effects of RYGB and its effects on normal metabolism, we investigated the effect of RYGB in lean pigs, using sham-operated pair-fed pigs as controls. Two weeks post-surgery, pigs were subjected to an intravenous glucose tolerance test (IVGTT) and circulating metabolites, hormones and lipids measured. Bile acid composition was profiled after extraction from blood, faeces and the gallbladder.

Results: A similar weight development in both groups of pigs validated our experimental model. Despite similar changes in fasting insulin, RYGB-pigs had lower fasting glucose levels. During an IVGTT RYGB-pigs had higher insulin and lower glucose levels. VLDL and IDL were lower in RYGB- than in sham-pigs. RYGB-pigs had increased levels of most amino acids, including branched-chain amino acids, but these were more efficiently suppressed by glucose. Levels of bile acids in the gallbladder were higher, whereas plasma and faecal bile acid levels were lower in RYGB- than in sham-pigs.

Conclusion: In a lean model RYGB caused lower plasma lipid and bile acid levels, which were compensated for by increased plasma amino acids, suggesting a switch from lipid to protein metabolism during fasting in the immediate postoperative period.

MeSH terms

  • Animals
  • Blood Glucose
  • Disease Models, Animal
  • Fasting / blood
  • Gastric Bypass / adverse effects*
  • Glucose / metabolism
  • Glucose Tolerance Test
  • Humans
  • Insulin / metabolism*
  • Insulin Resistance / physiology
  • Obesity / blood
  • Obesity / metabolism*
  • Obesity / physiopathology
  • Obesity / surgery*
  • Swine
  • Weight Loss / physiology

Substances

  • Blood Glucose
  • Insulin
  • Glucose

Grants and funding

This work was supported by grants from the Swedish Diabetes Foundation (PS, NW), the Royal Physiographic Society (PS), The Diabetes Wellness (NW), the Novo Nordisk Foundation (PS), Påhlsson (PS, NW), and Crafoord Foundations (PS), the Swedish Research Council incl. project grant (LG: Dnr. 521-2007-4037, NW: Dnr. 2008-4216, MG: Dnr. 2011-3900), strategic research area grant (EXODIAB: Dnr. 2009-1039) and Linnaeus grant (LUDC: Dnr. 349-2008-6589), an Advanced Research Grant from the European Research Council for (LG: GA 269045) as well as equipment grants from Wallenberg (KAW 2009-0243) and Lundberg foundation (grant nr. 359). In addition, the project was funded by the EU grant ENGAGE. The funders had no role in study design, data collection and analysis, decision to publish or preparation of the manuscript.