Bufalin reverses acquired drug resistance by inhibiting stemness in colorectal cancer cells

Oncol Rep. 2017 Sep;38(3):1420-1430. doi: 10.3892/or.2017.5826. Epub 2017 Jul 17.

Abstract

Drug resistance is an obstacle to chemotherapy in tumor patients. Recent studies have shown that the high stemness of cancer cells may be induced by chemotherapeutic drugs, which is correlated with drug resistance. In the present study, we investigated the effects of bufalin on the stemness of colorectal cancer. We found that cisplatin could induce high stemness through the tumorsphere formation assay in vitro and in vivo in the colorectal cancer cell lines HCT116 and LoVo. In addition, cisplatin-treated tumorsphere cells showed drug‑resistant properties. These results suggested that acquired drug resistance induced by cisplatin in colorectal cancer cells occurred via high stemness. On assessing the effects of bufalin, a traditional Chinese medicine monomer, we found that it could reverse the high stemness and drug resistance induced by cisplatin in colorectal cancer. These findings suggest that bufalin plays an adjuvant role in colorectal cancer chemotherapy and may help reverse acquired drug resistance. These findings may aid in the development of new therapeutic strategies.

MeSH terms

  • Antineoplastic Combined Chemotherapy Protocols / administration & dosage
  • Apoptosis / drug effects
  • Bufanolides / administration & dosage*
  • Cell Proliferation / drug effects
  • Cisplatin / administration & dosage
  • Cisplatin / adverse effects
  • Colorectal Neoplasms / drug therapy*
  • Colorectal Neoplasms / genetics
  • Colorectal Neoplasms / pathology
  • Drug Resistance, Neoplasm / drug effects*
  • HCT116 Cells
  • Humans
  • Neoplastic Stem Cells / drug effects*
  • Signal Transduction / drug effects

Substances

  • Bufanolides
  • Cisplatin
  • bufalin