Genetic variants in CYP2B6 and CYP2A6 explain interindividual variation in efavirenz plasma concentrations of HIV-infected children with diverse ethnic origin

PLoS One. 2017 Sep 8;12(9):e0181316. doi: 10.1371/journal.pone.0181316. eCollection 2017.

Abstract

Background: Approximately 2.6 million children live with HIV globally, and efavirenz (EFV) is one of the most widely used antiretroviral agents for HIV treatment in children and adults. There are concerns about the appropriateness of current EFV dosing and it has been discussed whether EFV dosing should be adapted according to genotype in children as suggested for adults.

Aim: To investigate if pediatric EFV dosing should be guided by genetic variation in drug metabolizing enzymes rather than by body weight.

Method: EFV plasma concentrations measured for clinical purposes from all children less than 18 years old at Karolinska University Hospital, Stockholm, Sweden, treated with EFV were collected retrospectively. They were genotyped for eleven polymorphisms in genes coding for drug-metabolizing enzymes and P-glycoprotein, of potential importance for EFV disposition. Data on country of origin, sex, age, weight, HIV RNA, viral resistance patterns, CD4 cells, adherence to treatment, subjective health status and adverse events were collected from their medical records.

Results: Thirty-six patients and 182 (mean 5 samples/patient) EFV plasma concentration measurements from children of African, Asian and Latin American origin were included. EFV plasma concentration varied 21-fold between measurements (n = 182) (0.85-19.3 mg/L) and 9-fold measured as mean EFV plasma concentration across the subjects (1.55-13.4 mg/L). A multivariate mixed-effects restricted maximum likelihood regression model, including multiple gene polymorphisms, identified CYP2B6*6 T/T (p < 0.0005), CYP2B6*11 G/G (p < 0.0005), CYP2A6*9 A/C (p = 0.001) genotypes, age at treatment initiation (p = 0.002) and time from treatment initiation (p < 0.0005) as independent factors significantly related to loge concentration/(dose/weight). The contribution of the model to the intra- and interindividual variation were 6 and 75%, respectively (Bryk/Raudenbush R-squared level).

Conclusion: Genetic polymorphisms in CYP2B6 and CYP2A6 explained a significant proportion of variability in EFV plasma concentration in HIV-infected children in a multi-ethnic outpatient clinic. Knowledge about individual variants in key drug metabolizing enzyme genes could improve clinical safety and genotype directed dosing could achieve more predictable EFV plasma concentrations in HIV-infected children.

MeSH terms

  • Age Factors
  • Alkynes
  • Alleles
  • Anti-HIV Agents / administration & dosage
  • Anti-HIV Agents / adverse effects
  • Anti-HIV Agents / pharmacokinetics*
  • Benzoxazines / administration & dosage
  • Benzoxazines / adverse effects
  • Benzoxazines / pharmacokinetics*
  • CD4 Lymphocyte Count
  • Child
  • Cyclopropanes
  • Cytochrome P-450 CYP2A6 / genetics*
  • Cytochrome P-450 CYP2B6 / genetics*
  • Ethnicity / genetics*
  • Female
  • Gene Frequency
  • Genotype
  • HIV / drug effects*
  • HIV Infections / drug therapy
  • HIV Infections / genetics*
  • HIV Infections / virology
  • Humans
  • Male
  • Pharmacogenomic Variants*
  • Polymorphism, Single Nucleotide
  • Sex Factors
  • Schweden
  • Viral Load

Substances

  • Alkynes
  • Anti-HIV Agents
  • Benzoxazines
  • Cyclopropanes
  • Cytochrome P-450 CYP2A6
  • Cytochrome P-450 CYP2B6
  • efavirenz

Grants and funding

The study was supported by grants from the Swedish Order of Freemason Foundation for Children (LN), the Samariten Foundation (SSA), the Jerring Foundation (LN), Karolinska Institutet Funds (2013tobi37895) (LN) and the Swedish Research Council (2011-3440 and VR-Link 2012-3466) (LLG). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.