Human Tissue-Resident Memory T Cells Are Defined by Core Transcriptional and Functional Signatures in Lymphoid and Mucosal Sites

Cell Rep. 2017 Sep 19;20(12):2921-2934. doi: 10.1016/j.celrep.2017.08.078.

Abstract

Tissue-resident memory T cells (TRMs) in mice mediate optimal protective immunity to infection and vaccination, while in humans, the existence and properties of TRMs remain unclear. Here, we use a unique human tissue resource to determine whether human tissue memory T cells constitute a distinct subset in diverse mucosal and lymphoid tissues. We identify a core transcriptional profile within the CD69+ subset of memory CD4+ and CD8+ T cells in lung and spleen that is distinct from that of CD69- TEM cells in tissues and circulation and defines human TRMs based on homology to the transcriptional profile of mouse CD8+ TRMs. Human TRMs in diverse sites exhibit increased expression of adhesion and inhibitory molecules, produce both pro-inflammatory and regulatory cytokines, and have reduced turnover compared with circulating TEM, suggesting unique adaptations for in situ immunity. Together, our results provide a unifying signature for human TRM and a blueprint for designing tissue-targeted immunotherapies.

Keywords: RNA-seq; human immunology; memory T cells; mucosal immunity.

MeSH terms

  • Animals
  • Antigens, CD / metabolism
  • Antigens, Differentiation, T-Lymphocyte / metabolism
  • Cell Lineage / genetics
  • Clone Cells
  • Gene Expression Profiling*
  • Humans
  • Immunologic Memory*
  • Lectins, C-Type / metabolism
  • Lymphocyte Activation / immunology
  • Lymphoid Tissue / immunology*
  • Mice
  • Mucous Membrane / immunology*
  • Phenotype
  • T-Lymphocytes / immunology*
  • Transcription, Genetic*
  • Transcriptome / genetics

Substances

  • Antigens, CD
  • Antigens, Differentiation, T-Lymphocyte
  • CD69 antigen
  • Lectins, C-Type