A Simple Method for Normalization of Aortic Contractility

J Vasc Res. 2018;55(3):177-186. doi: 10.1159/000490245. Epub 2018 Jul 5.

Abstract

Vascular contractile function changes in proliferative vascular diseases, e.g. atherosclerosis, and is documented using isolated blood vessels; yet, many laboratories differ in their approach to quantification. Some use raw values (e.g., mg, mN); others use a "percentage of control agonist" approach; and others normalize by blood vessel characteristic, e.g. length, mass, etc. A lack of uniformity limits direct comparison of contractility outcomes. To address this limitation, we developed a simple 2-step normalization method: (1) measure blood vessel segment length (mm), area (mm2) and calculate volume (mm3); then, (2) normalize isometric contraction (mN) by segment length and volume. Normalized aortic contractions but not raw values were statistically different between normal chow and high-fat diet-fed mice, supporting the practical utility and general applicability of normalization. It is recommended that aortic contractions be normalized to segment length and/or volume to reduce variability, enhance efficiency, and to foster universal comparisons across isometric myography platforms, laboratories, and experimental settings.

Keywords: Aorta; Blood vessel; Isometric contraction; Myography; Phenotypic switch; Vascular smooth muscle.

Publication types

  • Research Support, N.I.H., Extramural
  • Validation Study

MeSH terms

  • Animals
  • Aorta, Thoracic / drug effects
  • Aorta, Thoracic / pathology
  • Aorta, Thoracic / physiopathology*
  • Diet, High-Fat
  • Disease Models, Animal
  • Dose-Response Relationship, Drug
  • In Vitro Techniques
  • Isometric Contraction* / drug effects
  • Male
  • Mice, Inbred C57BL
  • Myography / methods
  • Myography / standards*
  • Proof of Concept Study
  • Rats, Transgenic
  • Receptors, LDL / deficiency
  • Receptors, LDL / genetics
  • Reproducibility of Results
  • Vasoconstriction* / drug effects
  • Vasoconstrictor Agents / pharmacology

Substances

  • Receptors, LDL
  • Vasoconstrictor Agents