Eosin treatment for psoriasis reduces skin leukocyte infiltration and secretion of inflammatory chemokines and angiogenic factors

Eur J Dermatol. 2018 Aug 1;28(4):457-466. doi: 10.1684/ejd.2018.3357.

Abstract

Background: Eosin has been traditionally employed as a topical treatment for psoriasis, but the biological mechanism of its therapeutic action has not been fully elucidated.

Objectives: To analyse eosin effects on psoriatic skin in vivo and keratinocytes and endothelial cells in vitro.

Materials & methods: Skin biopsies were taken from psoriatic plaques before and after a three-day eosin treatment and processed for histological analysis. Cultured human psoriatic keratinocytes and dermal endothelial cells were treated with eosin, and release of inflammatory chemokines was analysed by multiplexed bead-based immunoassay and ELISA.

Results: In patients, the three-day eosin treatment significantly reduced the number of infiltrating T lymphocytes, neutrophilic granulocytes, and dermal dendritic cells. A reduction in VEGF-A expression was also observed. In vitro, eosin treatment significantly decreased the release of CCL2, CCL5, and VEGF-A by keratinocytes and angiopoietin-2 by endothelial cells.

Conclusions: Eosin treatment impacts on psoriatic inflammatory infiltrates and dampens the release of proinflammatory chemokines and angiogenic factors.

Keywords: angiogenic growth factor; eosin; leucocyte; psoriasis.

MeSH terms

  • Angiopoietin-2 / metabolism
  • Cell Proliferation / drug effects
  • Cells, Cultured
  • Cytokines / metabolism
  • Dendritic Cells / immunology
  • Dermatologic Agents / pharmacology*
  • Dermatologic Agents / therapeutic use
  • Endothelial Cells / physiology
  • Eosine Yellowish-(YS) / pharmacology*
  • Eosine Yellowish-(YS) / therapeutic use
  • Humans
  • Keratinocytes / physiology
  • Neutrophil Infiltration
  • Psoriasis / drug therapy*
  • Psoriasis / immunology*
  • Psoriasis / metabolism
  • Psoriasis / pathology
  • T-Lymphocytes / immunology
  • Vascular Endothelial Growth Factor A / metabolism

Substances

  • Angiopoietin-2
  • Cytokines
  • Dermatologic Agents
  • Vascular Endothelial Growth Factor A
  • Eosine Yellowish-(YS)