[A case of Anderson-Fabry disease: a multidisciplinary approach for diagnosis and follow up]

G Ital Nefrol. 2018 Sep;35(5):2018-vol5.
[Article in Italian]

Abstract

Fabry disease (also known as Anderson-Fabry disease, angiocheratoma corporis diffusum, diffuse angiocheratoma) is a rare tesaurismosis linked to the deficiency of the lysosomal enzyme alpha-galactosidase A, required for the physiological catabolism of glycosphingolipids. The related clinical signs show a multisystemic feature and define a degenerative and disabling pathology, whose approach requires a close multidisciplinary specialist collaboration. Currently, the renewed interest in the disease is aimed at the need to provide an early diagnosis, in order to early begin the enzyme replacement therapy and to slow down or avoid the establishment of irreparable organ damage. For this reason, the diagnostic suspicion becomes crucial and arises from the careful observation and research of the symptoms, together with the anamnesis and the overall clinical evaluation of the patient.

Keywords: Fabry disease; alpha-galactosidase; enzymatic replacement therapy; sphingolipids.

Publication types

  • Case Reports

MeSH terms

  • Cerebral Hemorrhage / etiology
  • Early Diagnosis
  • Enzyme Replacement Therapy
  • Fabry Disease / complications
  • Fabry Disease / diagnosis*
  • Fabry Disease / drug therapy
  • Genetic Testing
  • Humans
  • Hypertrophy, Left Ventricular / etiology
  • Kidney Failure, Chronic / etiology
  • Kidney Failure, Chronic / therapy
  • Male
  • Middle Aged
  • Mutation, Missense
  • Pedigree
  • Point Mutation
  • alpha-Galactosidase / genetics
  • alpha-Galactosidase / therapeutic use

Substances

  • alpha-Galactosidase