Recombinant tumor necrosis factor can induce interleukin 2 receptor expression and cytolytic activity in a rat x mouse T cell hybrid

Eur J Immunol. 1987 Dec;17(12):1835-8. doi: 10.1002/eji.1830171224.

Abstract

Tumor necrosis factor (TNF), an endotoxin-induced macrophage monokine, is known for its cytotoxic and cytostatic effect on some tumor cell lines. Here we show that highly purified recombinant TNF, in combination with interleukin 2 (IL2), can induce IL2 receptor expression and cytolytic activity in a rat x mouse T cell hybrid (PC60). Previously, it was shown that IL1 had a similar effect on PC60 cells. The ability of TNF to co-induce IL2 receptor expression suggests that it may play a role in the activation of certain lymphoid effector cells. This observation augments the growing list of biological activities attributed to TNF.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cytotoxicity, Immunologic / drug effects*
  • Humans
  • Hybrid Cells
  • Immunity, Cellular / drug effects*
  • Interleukin-1 / pharmacology
  • Interleukin-2 / physiology*
  • Mice
  • Rats
  • Receptors, Immunologic / metabolism*
  • Receptors, Interleukin-2
  • Recombinant Proteins / pharmacology
  • T-Lymphocytes / immunology*
  • Tumor Necrosis Factor-alpha / pharmacology*

Substances

  • Interleukin-1
  • Interleukin-2
  • Receptors, Immunologic
  • Receptors, Interleukin-2
  • Recombinant Proteins
  • Tumor Necrosis Factor-alpha