Promoter-dependent nuclear RNA degradation ensures cell cycle-specific gene expression

Commun Biol. 2019 Jun 17:2:211. doi: 10.1038/s42003-019-0441-3. eCollection 2019.

Abstract

Cell cycle progression depends on phase-specific gene expression. Here we show that the nuclear RNA degradation machinery plays a lead role in promoting cell cycle-dependent gene expression by triggering promoter-dependent co-transcriptional RNA degradation. Single molecule quantification of RNA abundance in different phases of the cell cycle indicates that relative curtailment of gene expression in certain phases is attained even when transcription is not completely inhibited. When nuclear ribonucleases are deleted, transcription of the Saccharomyces cerevisiae G1-specific axial budding gene AXL2 is detected throughout the cell cycle and its phase-specific expression is lost. Promoter replacement abolished cell cycle-dependent RNA degradation and rendered the RNA insensitive to the deletion of nuclear ribonucleases. Together the data reveal a model of gene regulation whereby RNA abundance is controlled by promoter-dependent induction of RNA degradation.

Keywords: Mitosis; RNA decay.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Cycle*
  • Gene Expression Regulation, Fungal
  • Humans
  • Membrane Glycoproteins / genetics
  • Promoter Regions, Genetic*
  • RNA, Messenger / metabolism
  • RNA, Nuclear / metabolism*
  • Ribonuclease III / physiology
  • Saccharomyces cerevisiae Proteins / genetics
  • Saccharomyces cerevisiae Proteins / physiology
  • Transcription Factor TFIIA / genetics

Substances

  • Axl2 protein, S cerevisiae
  • Membrane Glycoproteins
  • RNA, Messenger
  • RNA, Nuclear
  • Saccharomyces cerevisiae Proteins
  • TOA1 protein, S cerevisiae
  • Transcription Factor TFIIA
  • RNT1 protein, S cerevisiae
  • Ribonuclease III

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