Abstract
Nonsteroidal anti-inflammatory drugs (NSAIDs) are among the most commonly used analgesics due to their lack of addictive potential. However, NSAIDs have the potential to cause serious gastrointestinal, renal, and cardiovascular adverse events. CYP2C9 polymorphisms influence metabolism and clearance of several drugs in this class, thereby affecting drug exposure and potentially safety. We summarize evidence from the published literature supporting these associations and provide therapeutic recommendations for NSAIDs based on CYP2C9 genotype (updates at www.cpicpgx.org).
© 2020 The Authors Clinical Pharmacology & Therapeutics © 2020 American Society for Clinical Pharmacology and Therapeutics.
Publication types
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Practice Guideline
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Research Support, N.I.H., Extramural
MeSH terms
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Anti-Inflammatory Agents, Non-Steroidal / adverse effects
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Anti-Inflammatory Agents, Non-Steroidal / pharmacokinetics*
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Clinical Decision-Making
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Consensus
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Cytochrome P-450 CYP2C9 / genetics*
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Cytochrome P-450 CYP2C9 / metabolism
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Drug Interactions
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Drug-Related Side Effects and Adverse Reactions / enzymology
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Drug-Related Side Effects and Adverse Reactions / genetics*
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Genotype
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Humans
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Pharmacogenetics / standards*
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Pharmacogenomic Testing / standards*
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Pharmacogenomic Variants*
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Phenotype
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Predictive Value of Tests
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Risk Assessment
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Risk Factors
Substances
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Anti-Inflammatory Agents, Non-Steroidal
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CYP2C9 protein, human
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Cytochrome P-450 CYP2C9