Calcium activity in response to nAChR ligands in murine bone marrow granulocytes with different Gr-1 expression

Cell Biol Int. 2021 Jul;45(7):1533-1545. doi: 10.1002/cbin.11593. Epub 2021 Apr 1.

Abstract

Polymorphonuclear neutrophilic granulocytes (PMNs) are the largest proportion of leukocytes in adult human blood that perform numerous functions, including phagocytosis, degranulation, generation of reactive oxygen species, and NETosis. Excessive neutrophil activity associates with hyperinflammation and tissue damage during pathologies such as inflammatory bowel disease, diabetes mellitus, tuberculosis, and coronavirus disease 2019. Nicotinic acetylcholine receptors (nAChRs) can modulate immune cells, including neutrophils, functions, therefore, nAChR ligands are considered as the potent agents for therapy of inflammation. Earlier it was shown, that about 30% of PMNs from the acute inflammatory site responded to nicotine by calcium spikes. In this study, we studied the generation of calcium spikes in murine granulocytes with different maturity level (evaluated by Gr-1 expression) isolated from bone marrow in response to ligands of nAChRs in control and under chronic nicotine consumption. It was found that nearly 20%-25% cells in the granulocyte population responded to nicotine or selective antagonists of different type of nAChRs (α-cobratoxin, GIC, and Vc1.1). We demonstrated that in the control group Ca2+ -mobilizing activity was regulated through α7 and α9α10 nAChRs in immature granulocytes (Gr-1int ), whereas in mature granulocytes (Gr-1hi ) it was regulated through α7, α3β2, and α9-contained nAChRs. Sensitivity of PMNs to nicotine depended on their maturity level after chronic nicotine consumption. Gr-1int cells responded to nicotine through α7 and α9-contained nAChRs, while Gr-1hi did not respond to nicotine. Thus, calcium response to nAChR ligands in bone marrow PMNs depends on their maturity level.

Keywords: Gr-1; asymmetry; calcium homoeostasis/signaling; immunity; ion channels; neutrophilic granulocytes; nicotinic acetylcholine receptors.

Publication types

  • Comparative Study

MeSH terms

  • Animals
  • Antigens, Ly / metabolism*
  • Bone Marrow Cells / drug effects*
  • Bone Marrow Cells / metabolism
  • Calcium / metabolism*
  • Calcium Signaling / drug effects*
  • Cells, Cultured
  • Cholinergic Agents / pharmacology*
  • Granulocytes / drug effects*
  • Granulocytes / metabolism
  • Ligands
  • Male
  • Mice
  • Mice, Inbred BALB C
  • Nicotine / pharmacology
  • Nicotinic Agonists / pharmacology
  • Nicotinic Antagonists / pharmacology
  • Receptors, Nicotinic / drug effects*
  • Receptors, Nicotinic / metabolism

Substances

  • Antigens, Ly
  • Cholinergic Agents
  • Ligands
  • Ly6G antigen, mouse
  • Nicotinic Agonists
  • Nicotinic Antagonists
  • Receptors, Nicotinic
  • Nicotine
  • Calcium