Retinoids Decrease Soluble MICA Concentration by Inhibiting the Enzymatic Activity of ADAM9 and ADAM10

Anticancer Res. 2021 May;41(5):2307-2320. doi: 10.21873/anticanres.15006.

Abstract

Background/aim: The association between MHC class I polypeptide-related sequence A (MICA) and hepatocellular carcinoma (HCC) development was identified in our previous genome-wide association study. Decreasing soluble MICA (sMICA) through MICA sheddases suppression facilitates natural killer (NK) cell-mediated cytotoxicity. The expression of ADAM9 in HCC has been correlated with poor prognosis, and our recent study showed that its suppression contributes to cancer elimination by decreasing sMICA.

Materials and methods: Human HCC cell line PLC/PRF/5 and HepG2 cells were used. sMICA levels were measured by ELISA. Expression of retinoid X receptors (RXRs) and retinoic acid receptors (RARs) was knocked down by siRNA.

Results: In our screening of FDA-approved drugs in vitro, retinoids were found to be efficient ADAM9 and ADAM10 inhibitors. Treatment with retinoids reduced sMICA levels in human HCC cells. Interestingly, the effects were abrogated by depletion of the retinoid receptor RXRα.

Conclusion: Retinoids can be potential novel agents for HCC treatment.

Keywords: ADAM10; ADAM9; HCC treatment; MICA; retinoids.

MeSH terms

  • ADAM Proteins / antagonists & inhibitors
  • ADAM Proteins / genetics
  • ADAM Proteins / metabolism*
  • ADAM10 Protein / antagonists & inhibitors
  • ADAM10 Protein / genetics
  • ADAM10 Protein / metabolism*
  • Biocatalysis / drug effects
  • Cell Line, Tumor
  • Cell Survival / drug effects
  • Drug Synergism
  • Hep G2 Cells
  • Histocompatibility Antigens Class I / genetics
  • Histocompatibility Antigens Class I / metabolism*
  • Humans
  • Membrane Proteins / antagonists & inhibitors
  • Membrane Proteins / genetics
  • Membrane Proteins / metabolism*
  • Molecular Structure
  • Phenylurea Compounds / pharmacology
  • Pyridines / pharmacology
  • RNA Interference
  • Retinoid X Receptors / genetics
  • Retinoid X Receptors / metabolism
  • Retinoids / chemistry
  • Retinoids / pharmacology*
  • Solubility

Substances

  • Histocompatibility Antigens Class I
  • MHC class I-related chain A
  • Membrane Proteins
  • Phenylurea Compounds
  • Pyridines
  • Retinoid X Receptors
  • Retinoids
  • regorafenib
  • ADAM Proteins
  • ADAM9 protein, human
  • ADAM10 Protein