Hypoxia enhances the hair growth-promoting effects of embryonic stem cell-derived mesenchymal stem cells via NADPH oxidase 4

Biomed Pharmacother. 2023 Mar:159:114303. doi: 10.1016/j.biopha.2023.114303. Epub 2023 Jan 25.

Abstract

Human embryonic stem cell (hES)-derived mesenchymal stem cells (-MSCs) are an unlimited source of MSCs. The hair growth-promoting effects of diverse MSCs have been reported, but not that of hES-MSCs. In the present study, we investigated the hair growth-promoting effects of hES-MSCs and their underlying mechanisms. hES-MSCs or conditioned medium of hES-MSCs exhibited hair-growth effects, which increased the length of mouse vibrissae and human hair follicles. hES-MSCs accelerated the telogen-to-anagen transition in C3H mice and were more effective than adipose-derived stem cells. We further examined whether hypoxia could enhance the hair-growth promoting effects of hES-MSCs. The injection of hES-MSCs or conditioned medium (Hyp-CM) cultured under hypoxia (2% O2) enhanced the telogen-to-anagen transition in C3H mice. Additionally, Hyp-CM increased the length of mouse vibrissae, human hair follicles, and the proliferation of human dermal papilla and outer root sheath cells. Moreover, fibroblast growth factor 7, interleukin 12B, and teratocarcinoma-derived growth factor 1 were upregulated under hypoxia, and the co-treatment with these three proteins increased the hair length and induced telogen-to-anagen transition. Hypoxia increased reactive oxygen species (ROS) production, and ROS scavenging attenuated the secretion of growth factors. NADPH oxidase 4 was primarily expressed in hES-MSCs and generated ROS under hypoxia. Collectively, our results suggest that hES-MSCs exhibit hair-growth effects, which is enhanced by hypoxia.

Keywords: Anagen induction; Embryonic stem cell-derived mesenchymal stem cells; Hair growth; Hypoxia; Reactive oxygen species.

MeSH terms

  • Animals
  • Cell Proliferation
  • Cells, Cultured
  • Culture Media, Conditioned / metabolism
  • Culture Media, Conditioned / pharmacology
  • Embryonic Stem Cells / metabolism
  • Hair Follicle* / metabolism
  • Humans
  • Hypoxia / metabolism
  • Intercellular Signaling Peptides and Proteins / metabolism
  • Mesenchymal Stem Cells* / metabolism
  • Mice
  • Mice, Inbred C3H
  • NADPH Oxidase 4 / metabolism
  • Reactive Oxygen Species / metabolism

Substances

  • NADPH Oxidase 4
  • Reactive Oxygen Species
  • Culture Media, Conditioned
  • Intercellular Signaling Peptides and Proteins