Epithelial-neuronal-immune cell interactions: Implications for immunity, inflammation, and tissue homeostasis at mucosal sites

J Allergy Clin Immunol. 2024 May;153(5):1169-1180. doi: 10.1016/j.jaci.2024.02.004. Epub 2024 Feb 16.

Abstract

The epithelial lining of the respiratory tract and intestine provides a critical physical barrier to protect host tissues against environmental insults, including dietary antigens, allergens, chemicals, and microorganisms. In addition, specialized epithelial cells communicate directly with hematopoietic and neuronal cells. These epithelial-immune and epithelial-neuronal interactions control host immune responses and have important implications for inflammatory conditions associated with defects in the epithelial barrier, including asthma, allergy, and inflammatory bowel diseases. In this review, we discuss emerging research that identifies the mechanisms and impact of epithelial-immune and epithelial-neuronal cross talk in regulating immunity, inflammation, and tissue homeostasis at mucosal barrier surfaces. Understanding the regulation and impact of these pathways could provide new therapeutic targets for inflammatory diseases at mucosal sites.

Keywords: Epithelium; airway inflammation; allergy; asthma; immune regulation; intestinal inflammation; mucosal immunity; neuroimmunity.

Publication types

  • Review
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Communication / immunology
  • Epithelial Cells* / immunology
  • Homeostasis* / immunology
  • Humans
  • Immunity, Mucosal
  • Inflammation* / immunology
  • Intestinal Mucosa / immunology
  • Mucous Membrane / immunology
  • Neurons* / immunology