Ketogenic diet induces p53-dependent cellular senescence in multiple organs

Sci Adv. 2024 May 17;10(20):eado1463. doi: 10.1126/sciadv.ado1463. Epub 2024 May 17.

Abstract

A ketogenic diet (KD) is a high-fat, low-carbohydrate diet that leads to the generation of ketones. While KDs improve certain health conditions and are popular for weight loss, detrimental effects have also been reported. Here, we show mice on two different KDs and, at different ages, induce cellular senescence in multiple organs, including the heart and kidney. This effect is mediated through adenosine monophosphate-activated protein kinase (AMPK) and inactivation of mouse double minute 2 (MDM2) by caspase-2, leading to p53 accumulation and p21 induction. This was established using p53 and caspase-2 knockout mice and inhibitors to AMPK, p21, and caspase-2. In addition, senescence-associated secretory phenotype biomarkers were elevated in serum from mice on a KD and in plasma samples from patients on a KD clinical trial. Cellular senescence was eliminated by a senolytic and prevented by an intermittent KD. These results have important clinical implications, suggesting that the effects of a KD are contextual and likely require individual optimization.

MeSH terms

  • AMP-Activated Protein Kinases / metabolism
  • Animals
  • Cellular Senescence*
  • Cyclin-Dependent Kinase Inhibitor p21 / genetics
  • Cyclin-Dependent Kinase Inhibitor p21 / metabolism
  • Diet, Ketogenic* / adverse effects
  • Mice
  • Mice, Knockout
  • Organ Specificity
  • Proto-Oncogene Proteins c-mdm2 / metabolism
  • Tumor Suppressor Protein p53* / genetics
  • Tumor Suppressor Protein p53* / metabolism

Substances

  • AMP-Activated Protein Kinases
  • Cyclin-Dependent Kinase Inhibitor p21
  • Proto-Oncogene Proteins c-mdm2
  • Tumor Suppressor Protein p53