Bioclay Enzyme with Bimetal Synergistic Sterilization and Infectious Wound Regeneration

Nano Lett. 2024 Jul 3;24(26):8046-8054. doi: 10.1021/acs.nanolett.4c01671. Epub 2024 Jun 24.

Abstract

Bacteria invasion is the main factor hindering the wound-healing process. However, current antibacterial therapies inevitably face complex challenges, such as the abuse of antibiotics or severe inflammation during treatment. Here, a drug-free bioclay enzyme (Bio-Clayzyme) consisting of Fe2+-tannic acid (TA) network-coated kaolinite nanoclay and glucose oxidase (GOx) was reported to destroy harmful bacteria via bimetal antibacterial therapy. At the wound site, Bio-Clayzyme was found to enhance the generation of toxic hydroxyl radicals for sterilization via cascade catalysis of GOx and Fe2+-mediated peroxidase mimetic activity. Specifically, the acidic characteristics of the infection microenvironment accelerated the release of Al3+ from kaolinite, which further led to bacterial membrane damage and amplified the antibacterial toxicity of Fe2+. Besides, Bio-Clayzyme also performed hemostasis and anti-inflammatory functions inherited from Kaol and TA. By the combination of hemostasis and anti-inflammatory and bimetal synergistic sterilization, Bio-Clayzyme achieves efficient healing of infected wounds, providing a revolutionary approach for infectious wound regeneration.

Keywords: anti-inflammation; bimetal synergistic sterilization; infected wound healing; infection microenvironment; nanoclay.

MeSH terms

  • Animals
  • Anti-Bacterial Agents* / chemistry
  • Anti-Bacterial Agents* / pharmacology
  • Clay / chemistry
  • Glucose Oxidase* / chemistry
  • Glucose Oxidase* / metabolism
  • Glucose Oxidase* / pharmacology
  • Iron / chemistry
  • Sterilization / methods
  • Wound Healing* / drug effects
  • Wound Infection / drug therapy
  • Wound Infection / microbiology

Substances

  • Glucose Oxidase
  • Anti-Bacterial Agents
  • Clay
  • Iron