Alveolar regeneration by airway secretory-cell-derived p63+ progenitors

Cell Stem Cell. 2024 Nov 7;31(11):1685-1700.e6. doi: 10.1016/j.stem.2024.08.005. Epub 2024 Sep 3.

Abstract

Lung injury activates epithelial stem or progenitor cells for alveolar repair and regeneration. Unraveling the origin and fate of injury-induced progenitors is crucial for elucidating lung repair mechanisms. Here, we report that p63-expressing progenitors emerge upon bleomycin-induced mouse lung injury. Single-cell RNA sequencing and clonal analysis reveal that these p63+ progenitors proliferate rapidly and differentiate into alveolar type 1 and type 2 cells through different trajectories. Dual recombinase-mediated sequential genetic-lineage tracing demonstrates that p63+ progenitors originate from airway secretory cells and subsequently generate alveolar cells. Functionally, p63 activation is essential for efficient alveolar regeneration from secretory cells post injury. Our study identifies secretory-cell-derived p63+ progenitors as contributors to alveolar repair, suggesting a potential therapeutic avenue for lung regeneration following injury.

Keywords: dual lineage tracing; lineage differentiation; lung stem cells; p63(+) progenitor; regeneration.

MeSH terms

  • Animals
  • Bleomycin / pharmacology
  • Cell Differentiation
  • Lung Injury / metabolism
  • Lung Injury / pathology
  • Mice
  • Mice, Inbred C57BL
  • Pulmonary Alveoli* / cytology
  • Pulmonary Alveoli* / metabolism
  • Regeneration*
  • Stem Cells* / cytology
  • Stem Cells* / metabolism
  • Trans-Activators / metabolism

Substances

  • Trp63 protein, mouse
  • Bleomycin
  • Trans-Activators