Co-regulation of innate and adaptive immune responses induced by ID93+GLA-SE vaccination in humans

Front Immunol. 2024 Sep 24:15:1441944. doi: 10.3389/fimmu.2024.1441944. eCollection 2024.

Abstract

Introduction: Development of an effective vaccine against tuberculosis is a critical step towards reducing the global burden of disease. A therapeutic vaccine might also reduce the high rate of TB recurrence and help address the challenges of drug-resistant strains. ID93+GLA-SE is a candidate subunit vaccine that will soon be evaluated in a phase 2b efficacy trial for prevention of recurrent TB among patients undergoing TB treatment. ID93+GLA-SE vaccination was shown to elicit robust CD4+ T cell and IgG antibody responses among recently treated TB patients in the TBVPX-203 Phase 2a study (NCT02465216), but the mechanisms underlying these responses are not well understood.

Methods: In this study we used specimens from TBVPX-203 participants to describe the changes in peripheral blood gene expression that occur after ID93+GLA-SE vaccination.

Results: Analyses revealed several distinct modules of co-varying genes that were either up- or down-regulated after vaccination, including genes associated with innate immune pathways at 3 days post-vaccination and genes associated with lymphocyte expansion and B cell activation at 7 days post-vaccination. Notably, the regulation of these gene modules was affected by the dose schedule and by participant sex, and early innate gene signatures were correlated with the ID93-specific CD4+ T cell response.

Discussion: The results provide insight into the complex interplay of the innate and adaptive arms of the immune system in developing responses to vaccination with ID93+GLA-SE and demonstrate how dosing and schedule can affect vaccine responses.

Keywords: RNA sequencing; innate; systems immunology; tuberculosis; vaccines.

Publication types

  • Clinical Trial, Phase II
  • Randomized Controlled Trial

MeSH terms

  • Adaptive Immunity*
  • Adult
  • Antibodies, Bacterial / blood
  • Antibodies, Bacterial / immunology
  • CD4-Positive T-Lymphocytes / immunology
  • Female
  • Humans
  • Immunity, Innate*
  • Male
  • Mycobacterium tuberculosis / immunology
  • Tuberculosis / immunology
  • Tuberculosis / prevention & control
  • Tuberculosis Vaccines* / administration & dosage
  • Tuberculosis Vaccines* / immunology
  • Vaccination*
  • Vaccines, Subunit / administration & dosage
  • Vaccines, Subunit / immunology

Substances

  • Tuberculosis Vaccines
  • Antibodies, Bacterial
  • Vaccines, Subunit

Grants and funding

The author(s) declare financial support was received for the research, authorship, and/or publication of this article. Funded in part by the Wellcome Trust (UNS31744) and with Federal funds from the National Institute of Allergy and Infectious Diseases, National Institutes of Health, Department of Health and Human Services, under the Seattle Tuberculosis Research Advancement Center (P30 AI168034) and Contract No. 75N93021C00029. The content is solely the responsibility of the authors and does not necessarily represent the official views of the National Institutes of Health.