Characterization of neuroendocrine differentiation in human benign prostate and prostatic adenocarcinoma

Cancer. 1993 Jun 15;71(12):3952-65. doi: 10.1002/1097-0142(19930615)71:12<3952::aid-cncr2820711226>3.0.co;2-x.

Abstract

Background: This report describes an immunohistopathologic analysis characterizing the incidence, pattern of distribution, and hormonal content of neuroendocrine (NE) cells in human benign prostate and prostatic adenocarcinoma.

Methods: Formaldehyde-fixed, paraffin-embedded material from 15 benign prostates, 31 primary prostatic adenocarcinomas, 16 metastatic lesions, 21 primary tumors treated with short-course diethylstilbestrol (DES), and 10 specimens from hormone-refractory patients were examined. NE cells were identified using silver histochemistry and a panel of immunohistochemical NE markers (chromogranin-A, serotonin, neuron-specific enolase), and specific peptide hormone antibodies.

Results: NE cells were identified in all benign prostates. NE cells were identified in 77% of primary untreated adenocarcinomas with no significant differences with respect to pathologic stage. NE cells were found isolated and dispersed in the tumor, composing the minority of malignant cells. Double-labeling and serial section immunohistochemistry demonstrated the coexpression of prostate-specific antigen (PSA) in NE cells. In addition to serotonin, some tumors expressed multiple hormone immunoreactivities. NE cells were identified in 56% of metastatic deposits, with a similar pattern of distribution. In DES-treated cases, NE cells were found consistently in the adjacent benign epithelium, whereas 52% of tumors contained NE cells. Hormone-refractory tumors contained NE cells in 60% of cases.

Conclusions: This analysis demonstrates that a significant proportion of primary and metastatic prostatic adenocarcinomas contain a subpopulation of NE cells, the expression of which does not appear to be suppressed with androgen ablation and does not correlate with pathologic stage. Furthermore, NE cells coexpress PSA, suggesting a common precursor cell of origin. The elaboration of biogenic amines and neuropeptides suggests that NE cells dispersed in prostatic carcinoma may play a paracrine growth-regulatory role.

MeSH terms

  • Adenocarcinoma / pathology*
  • Adenocarcinoma / secondary
  • Aged
  • Aged, 80 and over
  • Basement Membrane / pathology
  • Calcitonin / analysis
  • Carcinoma / drug therapy
  • Carcinoma / pathology
  • Cell Differentiation
  • Chromogranin A
  • Chromogranins / analysis
  • Cytoplasm / ultrastructure
  • Diethylstilbestrol / therapeutic use
  • Gastrin-Releasing Peptide
  • Gastrins / analysis
  • Humans
  • Keratins / analysis
  • Lymphatic Metastasis / pathology
  • Male
  • Middle Aged
  • Neoplasm Staging
  • Neurosecretory Systems / pathology*
  • Peptides / analysis
  • Prostate / pathology*
  • Prostate-Specific Antigen / analysis
  • Prostatic Neoplasms / drug therapy
  • Prostatic Neoplasms / pathology*
  • Seminal Vesicles / pathology
  • Serotonin / analysis
  • Staining and Labeling
  • Thyrotropin / analysis

Substances

  • Chromogranin A
  • Chromogranins
  • Gastrins
  • Peptides
  • Serotonin
  • Keratins
  • Diethylstilbestrol
  • Gastrin-Releasing Peptide
  • Thyrotropin
  • Calcitonin
  • Prostate-Specific Antigen