Functional commitment to helper T cell lineage precedes positive selection and is independent of T cell receptor MHC specificity

Immunity. 1994 Jul;1(4):269-76. doi: 10.1016/1074-7613(94)90078-7.

Abstract

Thymocyte differentiation proceeds from double positive CD4+CD8+ to single positive T cells. It has been proposed that this process occurs by an instructive or a stochastic mechanism. In this report, we show that in recombination-deficient mice (RAG-1-I-) constitutive expression of a CD8 transgene allows maturation of CD4+(CD8tg+) cells, which express mature levels of a transgenic class I-restricted T cell receptor, F5. Rescued F5+CD4+(CD8tg+) cells have equivalent levels of T cell receptor expression as CD8end+ cells, respond to cognate antigen and, upon stimulation, they exhibit a phenotype characteristic of CD4+ helper T cells. These data are consistent with a model of differentiation that predicts that thymocytes become functionally committed to a helper or cytotoxic lineage before the final step of positive selection and independently of MHC specificity of their T cell receptor.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • B-Lymphocytes / immunology
  • Base Sequence
  • CD4-Positive T-Lymphocytes / cytology
  • CD4-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / cytology
  • CD8-Positive T-Lymphocytes / immunology
  • Cell Differentiation
  • Cytotoxicity, Immunologic
  • DNA Primers / genetics
  • Genes, MHC Class I*
  • Homeodomain Proteins*
  • Interleukin-4 / biosynthesis
  • Interleukin-4 / genetics
  • Lymphocyte Activation
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Molecular Sequence Data
  • Peptides / immunology
  • Proteins / genetics
  • Proteins / metabolism
  • Receptors, Antigen, T-Cell / genetics*
  • T-Lymphocytes, Helper-Inducer / cytology
  • T-Lymphocytes, Helper-Inducer / immunology*

Substances

  • DNA Primers
  • Homeodomain Proteins
  • Peptides
  • Proteins
  • Receptors, Antigen, T-Cell
  • RAG-1 protein
  • Interleukin-4