Growth factor-induced stimulation of hexose transport in 3T3-L1 adipocytes: evidence that insulin-induced translocation of GLUT4 is independent of activation of MAP kinase

Cell Signal. 1994 Mar;6(3):313-20. doi: 10.1016/0898-6568(94)90036-1.

Abstract

We have examined the effect of growth factors on the rate of hexose transport in 3T3-L1 adipocytes. Epidermal growth factor (EGF) and platelet-derived growth factor (PDGF) were found to stimulate deoxyglucose transport by about 2-fold. The concentrations of EGF and PDGF which elicited half maximal responses were 100 and 350 pM, respectively. The increases in transport rate were acute effects; the stimulations were evident within minutes of exposure to growth factors. By contrast, insulin stimulated deoxyglucose transport approximately 16-fold over similar time periods. We have measured the appearance of both the insulin-responsive glucose transporter (GLUT4) and the erythrocyte-type glucose transporter (GLUT1) at the cell surface in response to insulin, EGF and PDGF. We show that both EGF and PDGF induce a 2-fold increase in GLUT1 at the cell surface, but both these growth factors were without effect on GLUT4 levels at the cell surface. In contrast, insulin induced a 13-fold increase in cell surface GLUT4. We further show that insulin, EGF and PDGF all activate MAP kinase as determined by a shift in electrophoretic mobility of this protein on SDS-PAGE. However, since the large translocation of GLUT4 to the cell surface is specific for insulin, we suggest that activation of MAP kinase is not the sole requisite for this process.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3T3 Cells
  • Adipocytes / metabolism*
  • Animals
  • Biological Transport / drug effects
  • Cells, Cultured
  • Deoxyglucose / metabolism*
  • Electrophoresis, Polyacrylamide Gel
  • Enzyme Activation / drug effects
  • Glucose Transporter Type 1
  • Glucose Transporter Type 4
  • Growth Substances / pharmacology*
  • Insulin / pharmacology*
  • Mice
  • Mitogen-Activated Protein Kinase 1
  • Monosaccharide Transport Proteins / metabolism*
  • Muscle Proteins*
  • Protein Serine-Threonine Kinases / metabolism*
  • Protein-Tyrosine Kinases / metabolism*

Substances

  • Glucose Transporter Type 1
  • Glucose Transporter Type 4
  • Growth Substances
  • Insulin
  • Monosaccharide Transport Proteins
  • Muscle Proteins
  • Slc2a1 protein, mouse
  • Slc2a4 protein, mouse
  • Deoxyglucose
  • Protein-Tyrosine Kinases
  • Protein Serine-Threonine Kinases
  • Mitogen-Activated Protein Kinase 1