Plasma amyloid precursor protein is decreased in Alzheimer's disease

Neuroreport. 1993 Jun;4(6):757-9. doi: 10.1097/00001756-199306000-00040.

Abstract

Alzheimer's disease is characterized by amyloid deposits whose major protein component is beta A4. beta A4 is a product of the amyloid precursor protein (APP). APP was assayed in partially purified plasma samples from 16 sporadic Alzheimer's disease patients, 12 age-matched controls, 15 Down's syndrome individuals aged 19-36 years and 8 young to middle-aged controls (22-51 years). 14 of the 16 Alzheimer's disease patients had decreased plasma APP when compared with age-matched controls. 14 of the 15 Down's syndrome individuals had similar levels of APP when compared with age-matched and elderly non-demented controls by immunoblotting, whereas one had levels of APP less than controls. Taken together with results from a previous report (Lancet 1992; 340: 453-454), the decreased plasma APP levels mirror the changes observed with cerebrospinal fluid APP levels in Alzheimer's disease.

MeSH terms

  • Adult
  • Alzheimer Disease / blood
  • Alzheimer Disease / metabolism*
  • Amyloid beta-Protein Precursor / blood
  • Amyloid beta-Protein Precursor / metabolism*
  • Blood Platelets / metabolism
  • Blotting, Western
  • Brain Chemistry / physiology
  • Down Syndrome / blood
  • Electrophoresis, Polyacrylamide Gel
  • Humans
  • Middle Aged

Substances

  • Amyloid beta-Protein Precursor