Presence of strA-strB gene within a streptomycin-resistance operon in a clinical isolate of Shigella flexneri

Pathology. 1996 Nov;28(4):356-8. doi: 10.1080/00313029600169344.

Abstract

Three aminoglycoside-modifying enzymes (AMEs) were produced by a clinical isolate of Shigella flexneri which was resistant to gentamicin, tobramycin, netilmicin, kanamycin, sisomicin and streptomycin: acetyltransferase (AAC) (3)-II-type, and phosphotransferase (APH) (3")- and (6)-type enzymes. The aminoglycoside-resistance genes were located on a 75-Kb plasmid. Two genes, strA-HK and strB-HK, in a transcriptional unit were found to code for streptomycin-resistance. The genetic organization and sequence of this transcriptional unit were identical to those of strA and strB in plasmid RSF1010. strA-HK and strB-HK when expressed separately produced functional enzymes. Our substrate profile study on the crude extracts of StrA-HK and StrB-HK proteins confirmed that StrA-HK was an APH(3")-type and showed that StrB-HK was a member of the APH(6) family.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Drug Resistance, Microbial / genetics
  • Operon / genetics
  • Phosphotransferases (Alcohol Group Acceptor) / genetics*
  • Restriction Mapping
  • Sequence Homology, Amino Acid
  • Shigella flexneri / genetics*
  • Streptomycin*

Substances

  • Phosphotransferases (Alcohol Group Acceptor)
  • streptomycin 6-kinase
  • streptomycin 3''-kinase
  • Streptomycin