Colonization by Streptococcus pneumoniae among human immunodeficiency virus-infected adults: prevalence of antibiotic resistance, impact of immunization, and characterization by polymerase chain reaction with BOX primers of isolates from persistent S. pneumoniae carriers

J Infect Dis. 1997 Mar;175(3):590-7. doi: 10.1093/infdis/175.3.590.

Abstract

Pharyngeal colonization by Streptococcus pneumoniae was evaluated in 103 human immunodeficiency virus (HIV)-infected subjects (<200 CD4 cells/microL, 57; > or = 200 CD4 cells/microL, 46) and 39 non-HIV-infected controls who were participants in a vaccine study. At baseline, 7%, 20%, and 10% of subjects in the <200 and > or = 200 CD4 cell groups and in the control group were colonized with S. pneumoniae: Rates at 6 months were 23%, 22%, and 0%, respectively. Of 34 isolates from HIV-infected subjects, 25 were penicillin-resistant and 19 were resistant to > or = 3 antimicrobials; of 8 isolates from controls, 1 was resistant. Resistance to trimethoprim-sulfamethoxazole was significantly higher among HIV-infected subjects with <200 CD4 cells/microL than in those with more CD4 cells. Polymerase chain reaction DNA analysis with BOX primers demonstrated that 12 HIV-infected subjects were persistently colonized with the same S. pneumoniae strain for > or = 1 month compared with none of the controls. HIV-infected subjects were more likely to be persistent pneumococcal carriers and to carry antibiotic-resistant isolates than were non-HIV-infected subjects.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial

MeSH terms

  • Adult
  • CD4 Lymphocyte Count
  • Carrier State
  • DNA, Bacterial / analysis
  • Drug Resistance, Microbial
  • HIV Infections / complications
  • HIV Infections / microbiology*
  • Humans
  • Male
  • Middle Aged
  • Pharyngitis / microbiology*
  • Pharynx / microbiology
  • Polymerase Chain Reaction
  • Risk Factors
  • Streptococcal Infections / complications*
  • Streptococcal Infections / microbiology
  • Streptococcus pneumoniae / pathogenicity*

Substances

  • DNA, Bacterial