Mechanism of gamma sigma T-cell-mediated inhibition of stem cell differentiation in vitro: possible relevance for myelosuppression in HIV-infected individuals

Cell Immunol. 1998 Feb 25;184(1):26-36. doi: 10.1006/cimm.1998.1257.

Abstract

We investigated whether gamma delta T cells contribute to the suppression of myelopoiesis in HIV infection. Freshly isolated gamma delta T cells from HIV seropositive patients suppressed CFU-GM growth in vitro. Preactivation of gamma delta T cells with IL-2 and/or IL-15 further reduced the number of CFU-GM. Natural killer cells and to a lower extent CD4+ and CD8+ cells also inhibited CFU-GM growth. In contrast to gamma delta T cells, this effect was not dependent on IL-15 or IL-2 preactivation. Moreover, no enhanced inhibitory effect of CD56+ and CD4+ cells was observed in HIV+ subjects compared to HIV- donors. The myelosuppressive effect of supernatants of gamma delta T cells could be inhibited by antibodies against IFN-gamma or TNF-alpha. Accordingly, we found increased numbers of TNF-alpha or IFN-gamma-secreting CD8+ gamma delta T cells in HIV+ patients. We conclude that the increased fraction of activated gamma delta T cells producing myelosuppressive cytokines might contribute to the dyshematopoiesis frequently observed in HIV-infected individuals.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Differentiation
  • HIV Infections / blood*
  • Hematopoietic Stem Cells / physiology*
  • Humans
  • Interferon-gamma / physiology
  • Interleukin-15 / pharmacology
  • Interleukin-2 / pharmacology
  • Leukopoiesis*
  • Receptors, Antigen, T-Cell, alpha-beta / physiology*
  • T-Lymphocytes / physiology*
  • Tumor Necrosis Factor-alpha / physiology

Substances

  • Interleukin-15
  • Interleukin-2
  • Receptors, Antigen, T-Cell, alpha-beta
  • Tumor Necrosis Factor-alpha
  • Interferon-gamma