Abstract
TAL1 gene deregulation is frequent in T-cell acute lymphoblastic leukemia (T-ALL) and can result from translocations involving 1p32 or, more frequently, from a cytogenetically undetectable interstitial deletion of chromosome 1. This study presents a case of T-ALL with a t(1;5)(p32;q31) involving TAL1, in which the breakpoint occurs approximately 10kbp 5' to the gene and leads to transcriptional activation and synthesis of a TAL1 protein, and extends the spectrum of recognized TAL1 gene translocations associated with T-ALL.
Publication types
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Case Reports
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Research Support, Non-U.S. Gov't
MeSH terms
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Adult
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Basic Helix-Loop-Helix Transcription Factors
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Chromosomes, Human, Pair 1 / genetics*
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Chromosomes, Human, Pair 5 / genetics*
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DNA, Neoplasm / analysis
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DNA, Neoplasm / genetics
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DNA, Neoplasm / isolation & purification
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DNA-Binding Proteins / genetics*
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Gene Expression / genetics
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Gene Expression Regulation, Neoplastic
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Humans
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Karyotyping
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Leukemia-Lymphoma, Adult T-Cell / genetics*
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Male
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Proto-Oncogene Proteins / genetics
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RNA, Messenger / analysis
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RNA, Messenger / genetics
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T-Cell Acute Lymphocytic Leukemia Protein 1
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Transcription Factors*
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Translocation, Genetic*
Substances
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Basic Helix-Loop-Helix Transcription Factors
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DNA, Neoplasm
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DNA-Binding Proteins
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Proto-Oncogene Proteins
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RNA, Messenger
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T-Cell Acute Lymphocytic Leukemia Protein 1
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Transcription Factors
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TAL1 protein, human