Hormonal regulation of microsomal flavin-containing monooxygenase activity by sex steroids and growth hormone in co-cultured adult male rat hepatocytes

Biochem Pharmacol. 1998 Oct 15;56(8):1047-51. doi: 10.1016/s0006-2952(98)00104-x.

Abstract

To investigate the hormonal control of the expression of flavin-containing monooxygenase (FMO; EC 1.14.13.8) under defined in vitro conditions, adult male rat hepatocytes were isolated by collagenase perfusion and co-cultured with rat liver epithelial cells of primitive biliary origin. The direct effect of 17beta-estradiol, testosterone, 5alpha-dihydrotestosterone (5alpha-DHT) and human growth hormone (hGH) on FMO activity was studied using this in vitro model. Optimal, non-cytotoxic hormonal concentrations were determined by measuring the lactate dehydrogenase (LDH) index. In addition, the microsomal protein content of the cultured hepatocytes was determined as a function of culture time. The female sex hormone 17beta-estradiol caused a significant decrease in FMO as a function of culture time. After 14 days of exposure, FMO activity decreased by 56%. Neither of the male sex hormones or human growth hormone had an effect on FMO activity. These results in co-cultured male rat hepatocytes support in vivo observation that 17beta-estradiol is a potent hormone involved in the negative regulation of the expression of FMO in male rat liver.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Coculture Techniques
  • Dihydrotestosterone / pharmacology
  • Epithelial Cells / drug effects
  • Estradiol / pharmacology
  • Gonadal Steroid Hormones / pharmacology*
  • Human Growth Hormone / pharmacology*
  • Liver / cytology
  • Liver / drug effects*
  • Liver / enzymology
  • Male
  • Microsomes, Liver / drug effects*
  • Microsomes, Liver / enzymology
  • Oxygenases / metabolism*
  • Rats
  • Rats, Sprague-Dawley
  • Sexual Maturation / physiology
  • Steroids / pharmacology*
  • Testosterone / pharmacology

Substances

  • Gonadal Steroid Hormones
  • Steroids
  • Dihydrotestosterone
  • Human Growth Hormone
  • Testosterone
  • Estradiol
  • Oxygenases
  • dimethylaniline monooxygenase (N-oxide forming)