Hayashidani S - Search Results

1

Direct evidence for increased hydroxyl radicals originating from superoxide in the failing myocardium.

Ide T, Tsutsui H, Kinugawa S, Suematsu N, Hayashidani S, Ichikawa K, Utsumi H, Machida Y, Egashira K, Takeshita A. Ide T, et al. Among authors: hayashidani s. Circ Res. 2000 Feb 4;86(2):152-7. doi: 10.1161/01.res.86.2.152. Circ Res. 2000. PMID: 10666410

2

Treatment with dimethylthiourea prevents left ventricular remodeling and failure after experimental myocardial infarction in mice: role of oxidative stress.

Kinugawa S, Tsutsui H, Hayashidani S, Ide T, Suematsu N, Satoh S, Utsumi H, Takeshita A. Kinugawa S, et al. Among authors: hayashidani s. Circ Res. 2000 Sep 1;87(5):392-8. doi: 10.1161/01.res.87.5.392. Circ Res. 2000. PMID: 10969037

3

Greater susceptibility of failing cardiac myocytes to oxygen free radical-mediated injury.

Tsutsui H, Ide T, Hayashidani S, Suematsu N, Utsumi H, Nakamura R, Egashira K, Takeshita A. Tsutsui H, et al. Among authors: hayashidani s. Cardiovasc Res. 2001 Jan;49(1):103-9. doi: 10.1016/s0008-6363(00)00197-8. Cardiovasc Res. 2001. PMID: 11121801

4

Positive inotropic effects of calcium sensitizers on normal and failing cardiac myocytes.

Tsutsui H, Kinugawa S, Ide T, Hayashidani S, Suematsu N, Satoh S, Nakamura R, Egashira K, Takeshita A. Tsutsui H, et al. Among authors: hayashidani s. J Cardiovasc Pharmacol. 2001 Jan;37(1):16-24. doi: 10.1097/00005344-200101000-00003. J Cardiovasc Pharmacol. 2001. PMID: 11152370

5

Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarction.

Ide T, Tsutsui H, Hayashidani S, Kang D, Suematsu N, Nakamura K, Utsumi H, Hamasaki N, Takeshita A. Ide T, et al. Among authors: hayashidani s. Circ Res. 2001 Mar 16;88(5):529-35. doi: 10.1161/01.res.88.5.529. Circ Res. 2001. PMID: 11249877

6

Effects of ACE inhibition on left ventricular failure and oxidative stress in Dahl salt-sensitive rats.

Tsutsui H, Ide T, Hayashidani S, Kinugawa S, Suematsu N, Utsumi H, Takeshita A. Tsutsui H, et al. Among authors: hayashidani s. J Cardiovasc Pharmacol. 2001 Jun;37(6):725-33. doi: 10.1097/00005344-200106000-00010. J Cardiovasc Pharmacol. 2001. PMID: 11392469

7

Enhanced generation of reactive oxygen species in the limb skeletal muscles from a murine infarct model of heart failure.

Tsutsui H, Ide T, Hayashidani S, Suematsu N, Shiomi T, Wen J, Nakamura Ki, Ichikawa K, Utsumi H, Takeshita A. Tsutsui H, et al. Among authors: hayashidani s. Circulation. 2001 Jul 10;104(2):134-6. doi: 10.1161/01.cir.104.2.134. Circulation. 2001. PMID: 11447074

8

Alpha1-adrenoceptor-Gq-RhoA signaling is upregulated to increase myofibrillar Ca2+ sensitivity in failing hearts.

Suematsu N, Satoh S, Kinugawa S, Tsutsui H, Hayashidani S, Nakamura R, Egashira K, Makino N, Takeshita A. Suematsu N, et al. Among authors: hayashidani s. Am J Physiol Heart Circ Physiol. 2001 Aug;281(2):H637-46. doi: 10.1152/ajpheart.2001.281.2.H637. Am J Physiol Heart Circ Physiol. 2001. PMID: 11454567 Free article.

9

8-oxo-dGTPase, which prevents oxidative stress-induced DNA damage, increases in the mitochondria from failing hearts.

Tsutsui H, Ide T, Shiomi T, Kang D, Hayashidani S, Suematsu N, Wen J, Utsumi H, Hamasaki N, Takeshita A. Tsutsui H, et al. Among authors: hayashidani s. Circulation. 2001 Dec 11;104(24):2883-5. doi: 10.1161/hc4901.101347. Circulation. 2001. PMID: 11739300

10

Fluvastatin, a 3-hydroxy-3-methylglutaryl coenzyme a reductase inhibitor, attenuates left ventricular remodeling and failure after experimental myocardial infarction.

Hayashidani S, Tsutsui H, Shiomi T, Suematsu N, Kinugawa S, Ide T, Wen J, Takeshita A. Hayashidani S, et al. Circulation. 2002 Feb 19;105(7):868-73. doi: 10.1161/hc0702.104164. Circulation. 2002. PMID: 11854129

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