Wohlford-Lenane C - Search Results

1

Airway inflammation and responsiveness in prostaglandin H synthase-deficient mice exposed to bacterial lipopolysaccharide.

Zeldin DC, Wohlford-Lenane C, Chulada P, Bradbury JA, Scarborough PE, Roggli V, Langenbach R, Schwartz DA. Zeldin DC, et al. Am J Respir Cell Mol Biol. 2001 Oct;25(4):457-65. doi: 10.1165/ajrcmb.25.4.4505. Am J Respir Cell Mol Biol. 2001. PMID: 11694451

2

Cytokine gene expression after inhalation of corn dust.

Wohlford-Lenane CL, Deetz DC, Schwartz DA. Wohlford-Lenane CL, et al. Am J Physiol. 1999 May;276(5):L736-43. doi: 10.1152/ajplung.1999.276.5.L736. Am J Physiol. 1999. PMID: 10330029

3

Variable airway responsiveness to inhaled lipopolysaccharide.

Kline JN, Cowden JD, Hunninghake GW, Schutte BC, Watt JL, Wohlford-Lenane CL, Powers LS, Jones MP, Schwartz DA. Kline JN, et al. Am J Respir Crit Care Med. 1999 Jul;160(1):297-303. doi: 10.1164/ajrccm.160.1.9808144. Am J Respir Crit Care Med. 1999. PMID: 10390415

4

IL-10 reduces grain dust-induced airway inflammation and airway hyperreactivity.

Quinn TJ, Taylor S, Wohlford-Lenane CL, Schwartz DA. Quinn TJ, et al. J Appl Physiol (1985). 2000 Jan;88(1):173-9. doi: 10.1152/jappl.2000.88.1.173. J Appl Physiol (1985). 2000. PMID: 10642378 Free article.

5

Endotoxin responsiveness and subchronic grain dust-induced airway disease.

George CL, Jin H, Wohlford-Lenane CL, O'Neill ME, Phipps JC, O'Shaughnessy P, Kline JN, Thorne PS, Schwartz DA. George CL, et al. Am J Physiol Lung Cell Mol Physiol. 2001 Feb;280(2):L203-13. doi: 10.1152/ajplung.2001.280.2.L203. Am J Physiol Lung Cell Mol Physiol. 2001. PMID: 11158998 Free article.

6

Inhibition of LPS-induced airway hyperresponsiveness and airway inflammation by LPS antagonists.

Schwartz DA, Christ WJ, Kleeberger SR, Wohlford-Lenane CL. Schwartz DA, et al. Am J Physiol Lung Cell Mol Physiol. 2001 Apr;280(4):L771-8. doi: 10.1152/ajplung.2001.280.4.L771. Am J Physiol Lung Cell Mol Physiol. 2001. PMID: 11238019 Free article.

7

Bacterial DNA or oligonucleotides containing unmethylated CpG motifs can minimize lipopolysaccharide-induced inflammation in the lower respiratory tract through an IL-12-dependent pathway.

Schwartz DA, Wohlford-Lenane CL, Quinn TJ, Krieg AM. Schwartz DA, et al. J Immunol. 1999 Jul 1;163(1):224-31. J Immunol. 1999. PMID: 10384120

8

TNF-alpha and IL-1 beta are not essential to the inflammatory response in LPS-induced airway disease.

Moreland JG, Fuhrman RM, Wohlford-Lenane CL, Quinn TJ, Benda E, Pruessner JA, Schwartz DA. Moreland JG, et al. Am J Physiol Lung Cell Mol Physiol. 2001 Jan;280(1):L173-80. doi: 10.1152/ajplung.2001.280.1.L173. Am J Physiol Lung Cell Mol Physiol. 2001. PMID: 11133507 Free article.

9

Spontaneous mutations in recombinant inbred mice: mutant toll-like receptor 4 (Tlr4) in BXD29 mice.

Cook DN, Whitehead GS, Burch LH, Berman KG, Kapadia Z, Wohlford-Lenane C, Schwartz DA. Cook DN, et al. Genetics. 2006 Mar;172(3):1751-5. doi: 10.1534/genetics.105.042820. Epub 2005 Dec 1. Genetics. 2006. PMID: 16322526 Free PMC article.

10

Genes other than TLR4 are involved in the response to inhaled LPS.

Lorenz E, Jones M, Wohlford-Lenane C, Meyer N, Frees KL, Arbour NC, Schwartz DA. Lorenz E, et al. Am J Physiol Lung Cell Mol Physiol. 2001 Nov;281(5):L1106-14. doi: 10.1152/ajplung.2001.281.5.L1106. Am J Physiol Lung Cell Mol Physiol. 2001. PMID: 11597901 Free article.

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