Baluda MA - Search Results

1

HPV-16 E6 oncoprotein impairs the fidelity of DNA end-joining via p53-dependent and -independent pathways.

Shin KH, Ahn JH, Kang MK, Lim PK, Yip FK, Baluda MA, Park NH. Shin KH, et al. Among authors: baluda ma. Int J Oncol. 2006 Jan;28(1):209-15. Int J Oncol. 2006. PMID: 16327998

2

Introduction of human telomerase reverse transcriptase to normal human fibroblasts enhances DNA repair capacity.

Shin KH, Kang MK, Dicterow E, Kameta A, Baluda MA, Park NH. Shin KH, et al. Among authors: baluda ma. Clin Cancer Res. 2004 Apr 1;10(7):2551-60. doi: 10.1158/1078-0432.ccr-0669-3. Clin Cancer Res. 2004. PMID: 15073136

3

Abnormal DNA end-joining activity in human head and neck cancer.

Shin KH, Kang MK, Kim RH, Kameta A, Baluda MA, Park NH. Shin KH, et al. Among authors: baluda ma. Int J Mol Med. 2006 May;17(5):917-24. Int J Mol Med. 2006. PMID: 16596281

4

Bmi-1 cooperates with human papillomavirus type 16 E6 to immortalize normal human oral keratinocytes.

Kim RH, Kang MK, Shin KH, Oo ZM, Han T, Baluda MA, Park NH. Kim RH, et al. Among authors: baluda ma. Exp Cell Res. 2007 Feb 1;313(3):462-72. doi: 10.1016/j.yexcr.2006.10.025. Epub 2006 Oct 28. Exp Cell Res. 2007. PMID: 17161394

5

p53 promotes the fidelity of DNA end-joining activity by, in part, enhancing the expression of heterogeneous nuclear ribonucleoprotein G.

Shin KH, Kim RH, Kang MK, Kim RH, Kim SG, Lim PK, Yochim JM, Baluda MA, Park NH. Shin KH, et al. Among authors: baluda ma. DNA Repair (Amst). 2007 Jun 1;6(6):830-40. doi: 10.1016/j.dnarep.2007.01.013. Epub 2007 Mar 26. DNA Repair (Amst). 2007. PMID: 17387044 Free PMC article.

6

HPV-16 oncogenes E6 and E7 are mutagenic in normal human oral keratinocytes.

Liu X, Han S, Baluda MA, Park NH. Liu X, et al. Among authors: baluda ma. Oncogene. 1997 May 15;14(19):2347-53. doi: 10.1038/sj.onc.1201078. Oncogene. 1997. PMID: 9178911

7

The telomeric length and heterogeneity decrease with age in normal human oral keratinocytes.

Kang MK, Swee J, Kim RH, Baluda MA, Park NH. Kang MK, et al. Among authors: baluda ma. Mech Ageing Dev. 2002 Mar 31;123(6):585-92. doi: 10.1016/s0047-6374(01)00407-9. Mech Ageing Dev. 2002. PMID: 11850022

8

Senescence-associated genes in normal human oral keratinocytes.

Kang MK, Kameta A, Shin KH, Baluda MA, Kim HR, Park NH. Kang MK, et al. Among authors: baluda ma. Exp Cell Res. 2003 Jul 15;287(2):272-81. doi: 10.1016/s0014-4827(03)00061-2. Exp Cell Res. 2003. PMID: 12837283

9

Telomere shortening does not occur during postmaturational aging in situ in normal human oral fibroblasts.

Kang MK, Kameta A, Baluda MA, Park NH. Kang MK, et al. Among authors: baluda ma. Mech Ageing Dev. 2003 Aug-Sep;124(8-9):873-6. doi: 10.1016/s0047-6374(03)00145-3. Mech Ageing Dev. 2003. PMID: 14499491

10

Senescence occurs with hTERT repression and limited telomere shortening in human oral keratinocytes cultured with feeder cells.

Kang MK, Kameta A, Shin KH, Baluda MA, Park NH. Kang MK, et al. Among authors: baluda ma. J Cell Physiol. 2004 Jun;199(3):364-70. doi: 10.1002/jcp.10410. J Cell Physiol. 2004. PMID: 15095283

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