Rossi MA - Search Results

1

Turbulent flow/low wall shear stress and stretch differentially affect aorta remodeling in rats.

Prado CM, Ramos SG, Alves-Filho JC, Elias J Jr, Cunha FQ, Rossi MA. Prado CM, et al. Among authors: rossi ma. J Hypertens. 2006 Mar;24(3):503-15. doi: 10.1097/01.hjh.0000209987.51606.23. J Hypertens. 2006. PMID: 16467654

2

Chronic inhibition of nitric oxide synthase induces hypertension and cardiomyocyte mitochondrial and myocardial collagen remodelling in the absence of hypertrophy.

Rossi MA, Ramos SG, Prado CM. Rossi MA, et al. J Hypertens. 2003 May;21(5):993-1001. doi: 10.1097/00004872-200305000-00025. J Hypertens. 2003. PMID: 12714875

3

Circumferential wall tension due to hypertension plays a pivotal role in aorta remodelling.

Prado CM, Rossi MA. Prado CM, et al. Among authors: rossi ma. Int J Exp Pathol. 2006 Dec;87(6):425-36. doi: 10.1111/j.1365-2613.2006.00506.x. Int J Exp Pathol. 2006. PMID: 17222210 Free PMC article.

4

Turbulent blood flow plays an essential localizing role in the development of atherosclerotic lesions in experimentally induced hypercholesterolaemia in rats.

Prado CM, Ramos SG, Elias J Jr, Rossi MA. Prado CM, et al. Among authors: rossi ma. Int J Exp Pathol. 2008 Feb;89(1):72-80. doi: 10.1111/j.1365-2613.2007.00564.x. Int J Exp Pathol. 2008. PMID: 18197872 Free PMC article.

5

Aorta remodeling responses to distinct atherogenic stimuli: hypertension, hypercholesterolemia and turbulent flow/low wall shear stress.

Prado CM, Rossi MA. Prado CM, et al. Among authors: rossi ma. Open Cardiovasc Med J. 2008;2:41-8. doi: 10.2174/1874192400802010041. Epub 2008 Jun 13. Open Cardiovasc Med J. 2008. PMID: 18949098 Free PMC article.

6

Imbalance between matrix metalloproteinases and tissue inhibitor of metalloproteinases in hypertensive vascular remodeling.

Castro MM, Rizzi E, Prado CM, Rossi MA, Tanus-Santos JE, Gerlach RF. Castro MM, et al. Among authors: rossi ma. Matrix Biol. 2010 Apr;29(3):194-201. doi: 10.1016/j.matbio.2009.11.005. Epub 2009 Dec 5. Matrix Biol. 2010. PMID: 19969080

7

Disruption of sarcolemmal dystrophin and beta-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis.

Celes MR, Torres-Dueñas D, Malvestio LM, Blefari V, Campos EC, Ramos SG, Prado CM, Cunha FQ, Rossi MA. Celes MR, et al. Among authors: rossi ma. Lab Invest. 2010 Apr;90(4):531-42. doi: 10.1038/labinvest.2010.3. Epub 2010 Feb 8. Lab Invest. 2010. PMID: 20142806 Free article.

8

Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins.

Celes MR, Torres-Dueñas D, Prado CM, Campos EC, Moreira JE, Cunha FQ, Rossi MA. Celes MR, et al. Among authors: rossi ma. Shock. 2010 Mar;33(3):322-31. doi: 10.1097/SHK.0b013e3181b38ef6. Shock. 2010. PMID: 20160610

9

Combining two potential causes of metalloproteinase secretion causes abdominal aortic aneurysms in rats: a new experimental model.

Mata KM, Prudente PS, Rocha FS, Prado CM, Floriano EM, Elias J Jr, Rizzi E, Gerlach RF, Rossi MA, Ramos SG. Mata KM, et al. Among authors: rossi ma. Int J Exp Pathol. 2011 Feb;92(1):26-39. doi: 10.1111/j.1365-2613.2010.00746.x. Epub 2010 Oct 29. Int J Exp Pathol. 2011. PMID: 21039990 Free PMC article.

10

Myocardial structural changes in long-term human severe sepsis/septic shock may be responsible for cardiac dysfunction.

Rossi MA, Celes MR, Prado CM, Saggioro FP. Rossi MA, et al. Shock. 2007 Jan;27(1):10-8. doi: 10.1097/01.shk.0000235141.05528.47. Shock. 2007. PMID: 17172974

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