Park NH - Search Results

1

Regulation of p53 during senescence in normal human keratinocytes.

Kim RH, Kang MK, Kim T, Yang P, Bae S, Williams DW, Phung S, Shin KH, Hong C, Park NH. Kim RH, et al. Among authors: park nh. Aging Cell. 2015 Oct;14(5):838-46. doi: 10.1111/acel.12364. Epub 2015 Jul 1. Aging Cell. 2015. PMID: 26138448 Free PMC article.

2

The telomeric length and heterogeneity decrease with age in normal human oral keratinocytes.

Kang MK, Swee J, Kim RH, Baluda MA, Park NH. Kang MK, et al. Among authors: park nh. Mech Ageing Dev. 2002 Mar 31;123(6):585-92. doi: 10.1016/s0047-6374(01)00407-9. Mech Ageing Dev. 2002. PMID: 11850022

3

Absence of hamTERT gene expression is associated with promoter hypermethylation in immortal hamster buccal pouch epithelial cells.

Kameta A, Kang MK, Shin KH, Park NH. Kameta A, et al. Among authors: park nh. Int J Oncol. 2003 Jun;22(6):1351-6. Int J Oncol. 2003. PMID: 12739004

4

Senescence-associated genes in normal human oral keratinocytes.

Kang MK, Kameta A, Shin KH, Baluda MA, Kim HR, Park NH. Kang MK, et al. Among authors: park nh. Exp Cell Res. 2003 Jul 15;287(2):272-81. doi: 10.1016/s0014-4827(03)00061-2. Exp Cell Res. 2003. PMID: 12837283

5

Telomere shortening does not occur during postmaturational aging in situ in normal human oral fibroblasts.

Kang MK, Kameta A, Baluda MA, Park NH. Kang MK, et al. Among authors: park nh. Mech Ageing Dev. 2003 Aug-Sep;124(8-9):873-6. doi: 10.1016/s0047-6374(03)00145-3. Mech Ageing Dev. 2003. PMID: 14499491

6

Introduction of human telomerase reverse transcriptase to normal human fibroblasts enhances DNA repair capacity.

Shin KH, Kang MK, Dicterow E, Kameta A, Baluda MA, Park NH. Shin KH, et al. Among authors: park nh. Clin Cancer Res. 2004 Apr 1;10(7):2551-60. doi: 10.1158/1078-0432.ccr-0669-3. Clin Cancer Res. 2004. PMID: 15073136

7

Senescence occurs with hTERT repression and limited telomere shortening in human oral keratinocytes cultured with feeder cells.

Kang MK, Kameta A, Shin KH, Baluda MA, Park NH. Kang MK, et al. Among authors: park nh. J Cell Physiol. 2004 Jun;199(3):364-70. doi: 10.1002/jcp.10410. J Cell Physiol. 2004. PMID: 15095283

8

Normal human oral keratinocytes demonstrate abnormal DNA end joining activity during replicative senescence.

Kang MK, Shin KH, Yip FK, Park NH. Kang MK, et al. Among authors: park nh. Mech Ageing Dev. 2005 Apr;126(4):475-9. doi: 10.1016/j.mad.2004.10.002. Epub 2004 Dec 9. Mech Ageing Dev. 2005. PMID: 15722106

9

Senescence-associated decline in the intranuclear accumulation of hOGG1-alpha and impaired 8-oxo-dG repair activity in senescing normal human oral keratinocytes in vivo.

Kang MK, Kim RH, Shin KH, Zhong W, Faull KF, Park NH. Kang MK, et al. Among authors: park nh. Exp Cell Res. 2005 Oct 15;310(1):186-95. doi: 10.1016/j.yexcr.2005.07.019. Exp Cell Res. 2005. PMID: 16122734

10

HPV-16 E6 oncoprotein impairs the fidelity of DNA end-joining via p53-dependent and -independent pathways.

Shin KH, Ahn JH, Kang MK, Lim PK, Yip FK, Baluda MA, Park NH. Shin KH, et al. Among authors: park nh. Int J Oncol. 2006 Jan;28(1):209-15. Int J Oncol. 2006. PMID: 16327998

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