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Interleukin-23 regulates interleukin-17 expression in wounds, and its inhibition accelerates diabetic wound healing through the alteration of macrophage polarization.
Lee J, Rodero MP, Patel J, Moi D, Mazzieri R, Khosrotehrani K. Lee J, et al. Among authors: patel j. FASEB J. 2018 Apr;32(4):2086-2094. doi: 10.1096/fj.201700773R. Epub 2018 Jan 5. FASEB J. 2018. PMID: 29208701
This study highlights the importance of the IL-17 pathway in wound closure offering new possibilities of therapeutic intervention in chronic wounds.-Lee, J., Rodero, M. P., Patel, J., Moi, D., Mazzieri, R., Khosrotehrani, K. Interleukin-23 regulates interleuk …
This study highlights the importance of the IL-17 pathway in wound closure offering new possibilities of therapeutic intervention in chronic …
A molecular classification of human mesenchymal stromal cells.
Rohart F, Mason EA, Matigian N, Mosbergen R, Korn O, Chen T, Butcher S, Patel J, Atkinson K, Khosrotehrani K, Fisk NM, Lê Cao KA, Wells CA. Rohart F, et al. Among authors: patel j. PeerJ. 2016 Mar 24;4:e1845. doi: 10.7717/peerj.1845. eCollection 2016. PeerJ. 2016. PMID: 27042394 Free PMC article.
Priming of endothelial colony-forming cells in a mesenchymal niche improves engraftment and vasculogenic potential by initiating mesenchymal transition orchestrated by NOTCH signaling.
Shafiee A, Patel J, Wong HY, Donovan P, Hutmacher DW, Fisk NM, Khosrotehrani K. Shafiee A, et al. Among authors: patel j. FASEB J. 2017 Feb;31(2):610-624. doi: 10.1096/fj.201600937. Epub 2016 Oct 24. FASEB J. 2017. PMID: 28045376
The collective results showed that primed ECFCs survive better and undergo a mesenchymal transition that is dependent on NOTCH signaling, resulting in significantly increased vasculogenic potential.-Shafiee, A., Patel, J., Wong, H. Y., Donovan, P., Hutmacher, D. W., …
The collective results showed that primed ECFCs survive better and undergo a mesenchymal transition that is dependent on NOTCH signaling, re …
6,290 results