Brandt SL - Search Results

1

Excessive localized leukotriene B4 levels dictate poor skin host defense in diabetic mice.

Brandt SL, Wang S, Dejani NN, Klopfenstein N, Winfree S, Filgueiras L, McCarthy BP, Territo PR, Serezani CH. Brandt SL, et al. JCI Insight. 2018 Sep 6;3(17):e120220. doi: 10.1172/jci.insight.120220. eCollection 2018 Sep 6. JCI Insight. 2018. PMID: 30185672 Free PMC article.

2

Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes.

Filgueiras LR, Brandt SL, Wang S, Wang Z, Morris DL, Evans-Molina C, Mirmira RG, Jancar S, Serezani CH. Filgueiras LR, et al. Among authors: brandt sl. Sci Signal. 2015 Jan 27;8(361):ra10. doi: 10.1126/scisignal.2005568. Sci Signal. 2015. PMID: 25628460 Free PMC article.

3

Topical Prostaglandin E Analog Restores Defective Dendritic Cell-Mediated Th17 Host Defense Against Methicillin-Resistant Staphylococcus Aureus in the Skin of Diabetic Mice.

Dejani NN, Brandt SL, Piñeros A, Glosson-Byers NL, Wang S, Son YM, Medeiros AI, Serezani CH. Dejani NN, et al. Among authors: brandt sl. Diabetes. 2016 Dec;65(12):3718-3729. doi: 10.2337/db16-0565. Epub 2016 Sep 7. Diabetes. 2016. PMID: 27605625 Free PMC article.

4

Too much of a good thing: How modulating LTB₄ actions restore host defense in homeostasis or disease.

Brandt SL, Serezani CH. Brandt SL, et al. Semin Immunol. 2017 Oct;33:37-43. doi: 10.1016/j.smim.2017.08.006. Semin Immunol. 2017. PMID: 29042027 Free PMC article. Review.

5

Macrophage-derived LTB4 promotes abscess formation and clearance of Staphylococcus aureus skin infection in mice.

Brandt SL, Klopfenstein N, Wang S, Winfree S, McCarthy BP, Territo PR, Miller L, Serezani CH. Brandt SL, et al. PLoS Pathog. 2018 Aug 13;14(8):e1007244. doi: 10.1371/journal.ppat.1007244. eCollection 2018 Aug. PLoS Pathog. 2018. PMID: 30102746 Free PMC article.

6

Leukotriene B₄ licenses inflammasome activation to enhance skin host defense.

Salina ACG, Brandt SL, Klopfenstein N, Blackman A, Bazzano JMR, Sá-Nunes A, Byers-Glosson N, Brodskyn C, Tavares NM, Da Silva IBS, Medeiros AI, Serezani CH. Salina ACG, et al. Among authors: brandt sl. Proc Natl Acad Sci U S A. 2020 Dec 1;117(48):30619-30627. doi: 10.1073/pnas.2002732117. Epub 2020 Nov 12. Proc Natl Acad Sci U S A. 2020. PMID: 33184178 Free PMC article.

7

SOCS-1 inhibition of type I interferon restrains Staphylococcus aureus skin host defense.

Klopfenstein N, Brandt SL, Castellanos S, Gunzer M, Blackman A, Serezani CH. Klopfenstein N, et al. Among authors: brandt sl. PLoS Pathog. 2021 Mar 10;17(3):e1009387. doi: 10.1371/journal.ppat.1009387. eCollection 2021 Mar. PLoS Pathog. 2021. PMID: 33690673 Free PMC article.

8

SOCS1 is a negative regulator of metabolic reprogramming during sepsis.

Piñeros Alvarez AR, Glosson-Byers N, Brandt S, Wang S, Wong H, Sturgeon S, McCarthy BP, Territo PR, Alves-Filho JC, Serezani CH. Piñeros Alvarez AR, et al. JCI Insight. 2017 Jul 6;2(13):e92530. doi: 10.1172/jci.insight.92530. eCollection 2017 Jul 6. JCI Insight. 2017. PMID: 28679957 Free PMC article.

9

Imbalance between HDAC and HAT activities drives aberrant STAT1/MyD88 expression in macrophages from type 1 diabetic mice.

Filgueiras LR, Brandt SL, Ramalho TR, Jancar S, Serezani CH. Filgueiras LR, et al. Among authors: brandt sl. J Diabetes Complications. 2017 Feb;31(2):334-339. doi: 10.1016/j.jdiacomp.2016.08.001. Epub 2016 Aug 7. J Diabetes Complications. 2017. PMID: 27623388 Free PMC article.

10

Nuclear PTEN enhances the maturation of a microRNA regulon to limit MyD88-dependent susceptibility to sepsis.

Sisti F, Wang S, Brandt SL, Glosson-Byers N, Mayo LD, Son YM, Sturgeon S, Filgueiras L, Jancar S, Wong H, Dela Cruz CS, Andrews N, Alves-Filho JC, Cunha FQ, Serezani CH. Sisti F, et al. Among authors: brandt sl. Sci Signal. 2018 May 1;11(528):eaai9085. doi: 10.1126/scisignal.aai9085. Sci Signal. 2018. PMID: 29717063 Free PMC article.

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