Haass C - Search Results

1

The case for low-level BACE1 inhibition for the prevention of Alzheimer disease.

McDade E, Voytyuk I, Aisen P, Bateman RJ, Carrillo MC, De Strooper B, Haass C, Reiman EM, Sperling R, Tariot PN, Yan R, Masters CL, Vassar R, Lichtenthaler SF. McDade E, et al. Among authors: haass c. Nat Rev Neurol. 2021 Nov;17(11):703-714. doi: 10.1038/s41582-021-00545-1. Epub 2021 Sep 21. Nat Rev Neurol. 2021. PMID: 34548654 Review.

2

Amyloid beta-peptide is produced by cultured cells during normal metabolism.

Haass C, Schlossmacher MG, Hung AY, Vigo-Pelfrey C, Mellon A, Ostaszewski BL, Lieberburg I, Koo EH, Schenk D, Teplow DB, et al. Haass C, et al. Nature. 1992 Sep 24;359(6393):322-5. doi: 10.1038/359322a0. Nature. 1992. PMID: 1383826

3

Mutation of the beta-amyloid precursor protein in familial Alzheimer's disease increases beta-protein production.

Citron M, Oltersdorf T, Haass C, McConlogue L, Hung AY, Seubert P, Vigo-Pelfrey C, Lieberburg I, Selkoe DJ. Citron M, et al. Among authors: haass c. Nature. 1992 Dec 17;360(6405):672-4. doi: 10.1038/360672a0. Nature. 1992. PMID: 1465129

4

Targeting of cell-surface beta-amyloid precursor protein to lysosomes: alternative processing into amyloid-bearing fragments.

Haass C, Koo EH, Mellon A, Hung AY, Selkoe DJ. Haass C, et al. Nature. 1992 Jun 11;357(6378):500-3. doi: 10.1038/357500a0. Nature. 1992. PMID: 1608449

5

The Swedish mutation causes early-onset Alzheimer's disease by beta-secretase cleavage within the secretory pathway.

Haass C, Lemere CA, Capell A, Citron M, Seubert P, Schenk D, Lannfelt L, Selkoe DJ. Haass C, et al. Nat Med. 1995 Dec;1(12):1291-6. doi: 10.1038/nm1295-1291. Nat Med. 1995. PMID: 7489411

6

Mutations associated with a locus for familial Alzheimer's disease result in alternative processing of amyloid beta-protein precursor.

Haass C, Hung AY, Selkoe DJ, Teplow DB. Haass C, et al. J Biol Chem. 1994 Jul 1;269(26):17741-8. J Biol Chem. 1994. PMID: 8021287 Free article.

7

Analysis of presenilin 1 and presenilin 2 expression and processing by newly developed monoclonal antibodies.

Diehlmann A, Ida N, Weggen S, Grünberg J, Haass C, Masters CL, Bayer TA, Beyreuther K. Diehlmann A, et al. Among authors: haass c. J Neurosci Res. 1999 May 15;56(4):405-19. doi: 10.1002/(SICI)1097-4547(19990515)56:4<405::AID-JNR8>3.0.CO;2-F. J Neurosci Res. 1999. PMID: 10340748

8

The presenilins in Alzheimer's disease--proteolysis holds the key.

Haass C, De Strooper B. Haass C, et al. Science. 1999 Oct 29;286(5441):916-9. doi: 10.1126/science.286.5441.916. Science. 1999. PMID: 10542139 Review.

9

Genes and mechanisms involved in beta-amyloid generation and Alzheimer's disease.

Steiner H, Capell A, Leimer U, Haass C. Steiner H, et al. Among authors: haass c. Eur Arch Psychiatry Clin Neurosci. 1999;249(6):266-70. doi: 10.1007/s004060050098. Eur Arch Psychiatry Clin Neurosci. 1999. PMID: 10653281 Review.

10

Presenilin-1 differentially facilitates endoproteolysis of the beta-amyloid precursor protein and Notch.

Capell A, Steiner H, Romig H, Keck S, Baader M, Grim MG, Baumeister R, Haass C. Capell A, et al. Among authors: haass c. Nat Cell Biol. 2000 Apr;2(4):205-11. doi: 10.1038/35008626. Nat Cell Biol. 2000. PMID: 10783238

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