Gebre-Medhin S - Search Results

1

Mice deficient for PDGF B show renal, cardiovascular, and hematological abnormalities.

Levéen P, Pekny M, Gebre-Medhin S, Swolin B, Larsson E, Betsholtz C. Levéen P, et al. Genes Dev. 1994 Aug 15;8(16):1875-87. doi: 10.1101/gad.8.16.1875. Genes Dev. 1994. PMID: 7958863 Free article.

2

PDGF-A signaling is a critical event in lung alveolar myofibroblast development and alveogenesis.

Boström H, Willetts K, Pekny M, Levéen P, Lindahl P, Hedstrand H, Pekna M, Hellström M, Gebre-Medhin S, Schalling M, Nilsson M, Kurland S, Törnell J, Heath JK, Betsholtz C. Boström H, et al. Cell. 1996 Jun 14;85(6):863-73. doi: 10.1016/s0092-8674(00)81270-2. Cell. 1996. PMID: 8681381 Free article.

3

Alveogenesis failure in PDGF-A-deficient mice is coupled to lack of distal spreading of alveolar smooth muscle cell progenitors during lung development.

Lindahl P, Karlsson L, Hellström M, Gebre-Medhin S, Willetts K, Heath JK, Betsholtz C. Lindahl P, et al. Development. 1997 Oct;124(20):3943-53. doi: 10.1242/dev.124.20.3943. Development. 1997. PMID: 9374392

4

Vascular endothelial growth factor-B-deficient mice display an atrial conduction defect.

Aase K, von Euler G, Li X, Pontén A, Thorén P, Cao R, Cao Y, Olofsson B, Gebre-Medhin S, Pekny M, Alitalo K, Betsholtz C, Eriksson U. Aase K, et al. Circulation. 2001 Jul 17;104(3):358-64. doi: 10.1161/01.cir.104.3.358. Circulation. 2001. PMID: 11457758

5

Increased insulin secretion and glucose tolerance in mice lacking islet amyloid polypeptide (amylin).

Gebre-Medhin S, Mulder H, Pekny M, Westermark G, Törnell J, Westermark P, Sundler F, Ahrén B, Betsholtz C. Gebre-Medhin S, et al. Biochem Biophys Res Commun. 1998 Sep 18;250(2):271-7. doi: 10.1006/bbrc.1998.9308. Biochem Biophys Res Commun. 1998. PMID: 9753619

6

Reduced nociceptive behavior in islet amyloid polypeptide (amylin) knockout mice.

Gebre-Medhin S, Mulder H, Zhang Y, Sundler F, Betsholtz C. Gebre-Medhin S, et al. Brain Res Mol Brain Res. 1998 Dec 10;63(1):180-3. doi: 10.1016/s0169-328x(98)00269-1. Brain Res Mol Brain Res. 1998. PMID: 9838101

7

Islet amyloid polypeptide--hen or egg in type 2 diabetes pathogenesis?

Betsholtz C, Christmanson L, Gebre-Medhin S, Westermark P. Betsholtz C, et al. Acta Oncol. 1993;32(2):149-54. doi: 10.3109/02841869309083904. Acta Oncol. 1993. PMID: 8323756 Review.

8

Targeted disruption of the mouse phospholipase C beta3 gene results in early embryonic lethality.

Wang S, Gebre-Medhin S, Betsholtz C, Stålberg P, Zhou Y, Larsson C, Weber G, Feinstein R, Oberg K, Gobl A, Skogseid B. Wang S, et al. FEBS Lett. 1998 Dec 18;441(2):261-5. doi: 10.1016/s0014-5793(98)01518-x. FEBS Lett. 1998. PMID: 9883896 Free article.

9

Islet amyloid polypeptide (amylin)-deficient mice develop a more severe form of alloxan-induced diabetes.

Mulder H, Gebre-Medhin S, Betsholtz C, Sundler F, Ahrén B. Mulder H, et al. Am J Physiol Endocrinol Metab. 2000 Apr;278(4):E684-91. doi: 10.1152/ajprenal.2000.278.4.F684. Am J Physiol Endocrinol Metab. 2000. PMID: 10751203 Free article.

10

Abolished tubuloglomerular feedback and increased plasma renin in adenosine A1 receptor-deficient mice.

Brown R, Ollerstam A, Johansson B, Skøtt O, Gebre-Medhin S, Fredholm B, Persson AE. Brown R, et al. Am J Physiol Regul Integr Comp Physiol. 2001 Nov;281(5):R1362-7. doi: 10.1152/ajpregu.2001.281.5.R1362. Am J Physiol Regul Integr Comp Physiol. 2001. PMID: 11641103 Free article.

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