Davies SW - Search Results

1

Transgenic mice in the study of polyglutamine repeat expansion diseases.

Bates GP, Mangiarini L, Davies SW. Bates GP, et al. Among authors: davies sw. Brain Pathol. 1998 Oct;8(4):699-714. doi: 10.1111/j.1750-3639.1998.tb00196.x. Brain Pathol. 1998. PMID: 9804379 Free PMC article. Review.

2

Are neuronal intranuclear inclusions the common neuropathology of triplet-repeat disorders with polyglutamine-repeat expansions?

Davies SW, Beardsall K, Turmaine M, DiFiglia M, Aronin N, Bates GP. Davies SW, et al. Lancet. 1998 Jan 10;351(9096):131-3. doi: 10.1016/S0140-6736(97)08360-8. Lancet. 1998. PMID: 9439509

3

Polyglutamine expansion and Huntington's disease.

Bates GP, Mangiarini L, Wanker EE, Davies SW. Bates GP, et al. Among authors: davies sw. Biochem Soc Trans. 1998 Aug;26(3):471-5. doi: 10.1042/bst0260471. Biochem Soc Trans. 1998. PMID: 9765898 Review. No abstract available.

4

Formation of polyglutamine inclusions in non-CNS tissue.

Sathasivam K, Hobbs C, Turmaine M, Mangiarini L, Mahal A, Bertaux F, Wanker EE, Doherty P, Davies SW, Bates GP. Sathasivam K, et al. Among authors: davies sw. Hum Mol Genet. 1999 May;8(5):813-22. doi: 10.1093/hmg/8.5.813. Hum Mol Genet. 1999. PMID: 10196370

5

Transgenic models of Huntington's disease.

Sathasivam K, Hobbs C, Mangiarini L, Mahal A, Turmaine M, Doherty P, Davies SW, Bates GP. Sathasivam K, et al. Among authors: davies sw. Philos Trans R Soc Lond B Biol Sci. 1999 Jun 29;354(1386):963-9. doi: 10.1098/rstb.1999.0447. Philos Trans R Soc Lond B Biol Sci. 1999. PMID: 10434294 Free PMC article. Review.

6

Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice.

Mangiarini L, Sathasivam K, Seller M, Cozens B, Harper A, Hetherington C, Lawton M, Trottier Y, Lehrach H, Davies SW, Bates GP. Mangiarini L, et al. Among authors: davies sw. Cell. 1996 Nov 1;87(3):493-506. doi: 10.1016/s0092-8674(00)81369-0. Cell. 1996. PMID: 8898202 Free article.

7

Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation.

Davies SW, Turmaine M, Cozens BA, DiFiglia M, Sharp AH, Ross CA, Scherzinger E, Wanker EE, Mangiarini L, Bates GP. Davies SW, et al. Cell. 1997 Aug 8;90(3):537-48. doi: 10.1016/s0092-8674(00)80513-9. Cell. 1997. PMID: 9267033 Free article.

8

Transgenic models of Huntington's disease.

Bates GP, Mangiarini L, Mahal A, Davies SW. Bates GP, et al. Among authors: davies sw. Hum Mol Genet. 1997;6(10):1633-7. doi: 10.1093/hmg/6.10.1633. Hum Mol Genet. 1997. PMID: 9300654 Review.

9

Transgenic mouse models of neurodegenerative disease caused by CAG/polyglutamine expansions.

Bates GP, Davies SW. Bates GP, et al. Among authors: davies sw. Mol Med Today. 1997 Nov;3(11):508-15. doi: 10.1016/S1357-4310(97)01142-8. Mol Med Today. 1997. PMID: 9430787 Review.

10

Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human huntington disease gene.

Cha JH, Kosinski CM, Kerner JA, Alsdorf SA, Mangiarini L, Davies SW, Penney JB, Bates GP, Young AB. Cha JH, et al. Among authors: davies sw. Proc Natl Acad Sci U S A. 1998 May 26;95(11):6480-5. doi: 10.1073/pnas.95.11.6480. Proc Natl Acad Sci U S A. 1998. PMID: 9600992 Free PMC article.

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