The association of diabetes with risk of prostate cancer defined by clinical and molecular features

Br J Cancer. 2020 Aug;123(4):657-665. doi: 10.1038/s41416-020-0910-y. Epub 2020 May 29.

Abstract

Background: To prospectively examine the association between diabetes and risk of prostate cancer defined by clinical and molecular features.

Methods: A total of 49,392 men from the Health Professionals Follow-up Study (HPFS) were followed from 1986 to 2014. Data on self-reported diabetes were collected at baseline and updated biennially. Clinical features of prostate cancer included localised, advanced, lethal, low-grade, intermediate-grade, and high-grade. Molecular features included TMPRSS2: ERG and PTEN subtypes. Cox proportional hazards regression models were used to evaluate the association between diabetes and incidence of subtype-specific prostate cancer.

Results: During 28 years of follow-up, we documented 6733 incident prostate cancer cases. Relative to men free from diabetes, men with diabetes had lower risks of total (HR: 0.82, 95% CI: 0.75-0.90), localised (HR: 0.82, 95% CI: 0.74-0.92), low-and intermediate-grade prostate cancer (HR: 0.77, 95% CI: 0.66-0.90; HR: 0.77, 95% CI: 0.65-0.91, respectively). For molecular subtypes, the HRs for ERG-negative and ERG-positive cases were 0.63 (0.42-0.95) and 0.72 (0.46-1.12); and for PTEN-intact and PTEN-loss cases were 0.69 (0.48-0.98) and 0.52 (0.19-1.41), respectively.

Conclusion: Besides providing advanced evidence for the inverse association between diabetes and prostate cancer, this study is the first to report associations between diabetes and ERG/PTEN defined prostate cancers.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Diabetes Mellitus / epidemiology*
  • Diabetes Mellitus / genetics
  • Follow-Up Studies
  • Humans
  • Incidence
  • Logistic Models
  • Male
  • Middle Aged
  • PTEN Phosphohydrolase / genetics*
  • Prospective Studies
  • Prostatic Neoplasms / epidemiology*
  • Prostatic Neoplasms / genetics
  • Prostatic Neoplasms / pathology
  • Serine Endopeptidases / genetics*
  • Transcriptional Regulator ERG / genetics

Substances

  • ERG protein, human
  • Transcriptional Regulator ERG
  • PTEN Phosphohydrolase
  • PTEN protein, human
  • Serine Endopeptidases
  • TMPRSS2 protein, human