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Cardiology Step 2 CK Notes
Cardiology Step 2 CK Notes
CONSTRICTIVE PERICARDITIS
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BRADYCARDIA
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AV block
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(Page 148).
MULTIFOCAL ATRIAL TACHYCARDIA
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ATRIAL FLUTTER
ETIOLOGY
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COPD
P embolism
Thyrotoxicosis
Mitral valve disease
Alcohol
ATRIAL FIBRILLATION
Cardiac conditions commonly assoc w the development of AF include rheumatic
mitral valve disease, coronary artery disease, CHF, and HTN (causes atrial
structures to dilate)
The standard of care is to slow the rate and anticoagulated IF the CHADS score is
<1. As a general concept, rate control alone is considered for the patient who
notices very little of the symptoms of the arrhythmia, while rhythm control is more
likely to be applied to the patient who immediately notices the arrhythmia and is
experiencing consequences of the arrhythmia, such as shortness of breath, or
development of heart failure.
Drugs proven effective for pharmacologic cardioversion of atrial fibrillation include:
amiodarone, dofetilide, flecainide, ibutilide, propafenone, and quinidine.
Drugs used to maintain sinus rhythm in patients with atrial fibrillation include
amiodarone, disopyramide, dofetilide, flecainide, propafenone, and
sotalol. Rate control is the standard of care for most patients.
The following drugs are recommended for their demonstrated efficacy in rate
control at rest and during exercise: diltiazem, atenolol, metoprolol, and verapamil.
Control the heart rate, then anticoagulate. Use aspirin for those with CHADS 0 or 1,
and dabigatran, rivaroxaban, or warfarin for CHADS 2 or more. Heparin is not
necessary prior to starting oral anticoagulants.
WOLFF-PARKINSON-WHITE SYNDROME
If the patient is hemodynamically stable, then procainamide is the best medication.
Avoid digoxin, and calcium-channel blockers; these medications can inhibit
conduction in the normal conduction pathway.
Ablation is the definitive treatment
VENTRICULAR FIBRILLATION
Arrhythmias initiated by a ventricular premature beat in the setting of abnormal
ventricular repolarization characterized by prolongation of the QT interval.
PULSELESS ARREST
AMIODARONE
The most severe side effects of amiodarone therapy are related to the lungs and
present as cough, fever, or painful breathing. These reactions can be fatal. About
20% of patients who receive amiodarone experience some form of nerve toxicity.
Symptoms may include imbalance or changes in gait, tremor, numbness in the
fingers or toes, dizziness, muscle weakness, or loss of coordination.