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CARDIOLOGY (INTERNAL MEDICINE) NOTES

CONSTRICTIVE PERICARDITIS
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BRADYCARDIA
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Depression of sinus automacity


o Hypothyroidism
o Swimming and marathon runner
Excessive vagal tone

AV block
-

May be classified anatomically based on the site of block as determined by


His Bundle EKG

FIRST DEGREE HEART BLOCK


Etiology
Degenerative changes in the AV conduction system caused by:
Aging
Digitalis
Exaggerated vagal tone
Ischemia (diaphragmatic infarction)
Inflammation (myocarditis, acute rheumatic fever)
Cardiomyopathies
SECOND DEGREE HEART BLOCK
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Mobitz 1 PR prolongation the dropped beat (causes degenerative changes


in AV node; diaphragmatic MI; digitalis toxicity; myocarditis; rheumatic fever;
increased vagal node)
Mobitz 2 no PR prolongation (causes extensive anterior myocardial infarct;
degenerative changes in His-Purkinje system; massive calcification of mitral
or aortic valve annulus)

THIRD DEGREE HEART BLOCK


Etiology
Most common cause in adults is simple fibrous degenerative changes in the
conduction system that results from aging (Lengre disease)
Inferior or posterior infarction
Infectious and inflammatory processes, such as abscesses, tubercles, tumors,
infiltrative disease of the myocardium, sarcoid nodules, and gummas,
myocarditis, and rheumatic fever
Drugs like digitalis
Ankylosing spondylitis

(Page 148).
MULTIFOCAL ATRIAL TACHYCARDIA
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Multifocal atrial tachycardia is characterized by an irregular supraventricular


rhythm, at rates 100200 beats/min.
The morphology of the P waves (at least 3 different P wave forms) varies
from beat to beat, as does the PR interval. Each QRS complex, however, is
preceded by a P wave.
Generally seen in elderly patients or those with chronic lung disease
who are experiencing respiratory failure
Use diltiazem, verapamil, or digoxin; avoid beta blockers because of lung
disease
(Page 149).

ATRIAL FLUTTER
ETIOLOGY
-

COPD
P embolism
Thyrotoxicosis
Mitral valve disease
Alcohol

ATRIAL FIBRILLATION
Cardiac conditions commonly assoc w the development of AF include rheumatic
mitral valve disease, coronary artery disease, CHF, and HTN (causes atrial
structures to dilate)

The standard of care is to slow the rate and anticoagulated IF the CHADS score is
<1. As a general concept, rate control alone is considered for the patient who
notices very little of the symptoms of the arrhythmia, while rhythm control is more
likely to be applied to the patient who immediately notices the arrhythmia and is
experiencing consequences of the arrhythmia, such as shortness of breath, or
development of heart failure.
Drugs proven effective for pharmacologic cardioversion of atrial fibrillation include:
amiodarone, dofetilide, flecainide, ibutilide, propafenone, and quinidine.
Drugs used to maintain sinus rhythm in patients with atrial fibrillation include
amiodarone, disopyramide, dofetilide, flecainide, propafenone, and
sotalol. Rate control is the standard of care for most patients.
The following drugs are recommended for their demonstrated efficacy in rate
control at rest and during exercise: diltiazem, atenolol, metoprolol, and verapamil.
Control the heart rate, then anticoagulate. Use aspirin for those with CHADS 0 or 1,
and dabigatran, rivaroxaban, or warfarin for CHADS 2 or more. Heparin is not
necessary prior to starting oral anticoagulants.
WOLFF-PARKINSON-WHITE SYNDROME
If the patient is hemodynamically stable, then procainamide is the best medication.
Avoid digoxin, and calcium-channel blockers; these medications can inhibit
conduction in the normal conduction pathway.
Ablation is the definitive treatment
VENTRICULAR FIBRILLATION
Arrhythmias initiated by a ventricular premature beat in the setting of abnormal
ventricular repolarization characterized by prolongation of the QT interval.
PULSELESS ARREST

AMIODARONE
The most severe side effects of amiodarone therapy are related to the lungs and
present as cough, fever, or painful breathing. These reactions can be fatal. About
20% of patients who receive amiodarone experience some form of nerve toxicity.
Symptoms may include imbalance or changes in gait, tremor, numbness in the
fingers or toes, dizziness, muscle weakness, or loss of coordination.

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